"Bcl-2-Like Protein 11" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A BCL-2-like protein that has a C-terminal BCL-2 homology (BH3) domain and forms heterodimers with other BCL-2 FAMILY PROTEINS. It is a strong inducer of APOPTOSIS and ANOIKIS; several isoforms are expressed (BimEL, Bim L, Bim-alpha, Bim-s; and Bim-gamma) that have different potencies for inducing apoptosis.
| Descriptor ID |
D000072224
|
| MeSH Number(s) |
D12.644.360.075.323 D12.776.476.075.323 D12.776.543.116 D12.776.624.664.700.025
|
| Concept/Terms |
Bcl-2-Like Protein 11- Bcl-2-Like Protein 11
- 11, Bcl-2-Like Protein
- Bcl 2 Like Protein 11
- Protein 11, Bcl-2-Like
- Bcl-2-Interacting Mediator of Cell Death
- Bcl 2 Interacting Mediator of Cell Death
- Bcl-2-Binding Protein, BIM
- BIM Bcl-2-Binding Protein
- Bcl 2 Binding Protein, BIM
- Protein, BIM Bcl-2-Binding
- BCL2L11 Protein
- Protein, BCL2L11
- BIM Protein
- Protein, BIM
|
Below are MeSH descriptors whose meaning is more general than "Bcl-2-Like Protein 11".
Below are MeSH descriptors whose meaning is more specific than "Bcl-2-Like Protein 11".
This graph shows the total number of publications written about "Bcl-2-Like Protein 11" by people in this website by year, and whether "Bcl-2-Like Protein 11" was a major or minor topic of these publications.
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Below are the most recent publications written about "Bcl-2-Like Protein 11" by people in Profiles.
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ZAP-70 Regulates Autoimmune Arthritis via Alterations in T Cell Activation and Apoptosis. Cells. 2019 05 24; 8(5).
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Pseudomonas aeruginosa ExoS Induces Intrinsic Apoptosis in Target Host Cells in a Manner That is Dependent on its GAP Domain Activity. Sci Rep. 2018 09 19; 8(1):14047.
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Pseudomonas aeruginosa ExoT Induces Mitochondrial Apoptosis in Target Host Cells in a Manner That Depends on Its GTPase-activating Protein (GAP) Domain Activity. J Biol Chem. 2015 Nov 27; 290(48):29063-73.