"Myocytes, Cardiac" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Striated muscle cells found in the heart. They are derived from cardiac myoblasts (MYOBLASTS, CARDIAC).
| Descriptor ID |
D032383
|
| MeSH Number(s) |
A07.541.704.570 A10.690.552.750.570 A11.620.500
|
| Concept/Terms |
Myocytes, Cardiac- Myocytes, Cardiac
- Cardiac Myocyte
- Cardiac Myocytes
- Myocyte, Cardiac
- Muscle Cells, Heart
- Cell, Heart Muscle
- Cells, Heart Muscle
- Heart Muscle Cell
- Heart Muscle Cells
- Muscle Cell, Heart
- Cardiomyocytes
- Cardiomyocyte
- Muscle Cells, Cardiac
- Cardiac Muscle Cell
- Cardiac Muscle Cells
- Cell, Cardiac Muscle
- Cells, Cardiac Muscle
- Muscle Cell, Cardiac
|
Below are MeSH descriptors whose meaning is more general than "Myocytes, Cardiac".
Below are MeSH descriptors whose meaning is more specific than "Myocytes, Cardiac".
This graph shows the total number of publications written about "Myocytes, Cardiac" by people in this website by year, and whether "Myocytes, Cardiac" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2002 | 2 | 0 | 2 |
| 2003 | 8 | 0 | 8 |
| 2004 | 6 | 3 | 9 |
| 2005 | 5 | 4 | 9 |
| 2006 | 7 | 0 | 7 |
| 2007 | 9 | 1 | 10 |
| 2008 | 7 | 1 | 8 |
| 2009 | 6 | 0 | 6 |
| 2010 | 6 | 1 | 7 |
| 2011 | 6 | 2 | 8 |
| 2012 | 8 | 2 | 10 |
| 2013 | 9 | 2 | 11 |
| 2014 | 8 | 1 | 9 |
| 2015 | 2 | 0 | 2 |
| 2016 | 5 | 0 | 5 |
| 2017 | 8 | 4 | 12 |
| 2018 | 3 | 1 | 4 |
| 2019 | 2 | 0 | 2 |
| 2020 | 4 | 0 | 4 |
| 2021 | 2 | 2 | 4 |
| 2022 | 2 | 3 | 5 |
| 2024 | 0 | 1 | 1 |
To return to the timeline,
click here.
Below are the most recent publications written about "Myocytes, Cardiac" by people in Profiles.
-
Role of Mitochondrial ROS for Calcium Alternans in Atrial Myocytes. Biomolecules. 2024 Jan 24; 14(2).
-
Long-Term Alcohol-Activated c-Jun N-terminal Kinase Isoform 2 Preserves Cardiac Function but Drives Ca2+-Triggered Arrhythmias. Cells. 2023 09 08; 12(18).
-
Renal Denervation Helps Preserve the Ejection Fraction by Preserving Endocardial-Endothelial Function during Heart Failure. Int J Mol Sci. 2023 Apr 15; 24(8).
-
The 'Reverse FDUF' Mechanism of Atrial Excitation-Contraction Coupling Sustains Calcium Alternans-A Hypothesis. Biomolecules. 2022 12 20; 13(1).
-
Activation of small conductance Ca2+ -activated K+ channels suppresses Ca2+ transient and action potential alternans in ventricular myocytes. J Physiol. 2023 01; 601(1):51-67.
-
The HVCN1 voltage-gated proton channel contributes to pH regulation in canine ventricular myocytes. J Physiol. 2022 05; 600(9):2089-2103.
-
Transthyretin deposition alters cardiomyocyte sarcomeric architecture, calcium transients, and contractile force. Physiol Rep. 2022 03; 10(5):e15207.
-
L-type Ca2+ channel recovery from inactivation in rabbit atrial myocytes. Physiol Rep. 2022 03; 10(5):e15222.
-
Ero1a-Dependent ERp44 Dissociation From RyR2 Contributes to Cardiac Arrhythmia. Circ Res. 2022 03 04; 130(5):711-724.
-
Inositol 1,4,5-trisphosphate receptor - reactive oxygen signaling domain regulates excitation-contraction coupling in atrial myocytes. J Mol Cell Cardiol. 2022 02; 163:147-155.