"Myocytes, Cardiac" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Striated muscle cells found in the heart. They are derived from cardiac myoblasts (MYOBLASTS, CARDIAC).
| Descriptor ID |
D032383
|
| MeSH Number(s) |
A07.541.704.570 A10.690.552.750.570 A11.620.500
|
| Concept/Terms |
Myocytes, Cardiac- Myocytes, Cardiac
- Cardiac Myocyte
- Cardiac Myocytes
- Myocyte, Cardiac
- Muscle Cells, Heart
- Cell, Heart Muscle
- Cells, Heart Muscle
- Heart Muscle Cell
- Heart Muscle Cells
- Muscle Cell, Heart
- Cardiomyocytes
- Cardiomyocyte
- Muscle Cells, Cardiac
- Cardiac Muscle Cell
- Cardiac Muscle Cells
- Cell, Cardiac Muscle
- Cells, Cardiac Muscle
- Muscle Cell, Cardiac
|
Below are MeSH descriptors whose meaning is more general than "Myocytes, Cardiac".
Below are MeSH descriptors whose meaning is more specific than "Myocytes, Cardiac".
This graph shows the total number of publications written about "Myocytes, Cardiac" by people in this website by year, and whether "Myocytes, Cardiac" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2003 | 4 | 0 | 4 |
| 2004 | 4 | 2 | 6 |
| 2005 | 1 | 3 | 4 |
| 2006 | 2 | 0 | 2 |
| 2007 | 5 | 0 | 5 |
| 2008 | 3 | 1 | 4 |
| 2009 | 4 | 0 | 4 |
| 2010 | 2 | 1 | 3 |
| 2011 | 2 | 1 | 3 |
| 2012 | 3 | 2 | 5 |
| 2013 | 6 | 2 | 8 |
| 2014 | 6 | 0 | 6 |
| 2016 | 3 | 0 | 3 |
| 2017 | 3 | 2 | 5 |
| 2018 | 3 | 1 | 4 |
| 2019 | 1 | 0 | 1 |
| 2020 | 1 | 0 | 1 |
| 2021 | 1 | 2 | 3 |
| 2022 | 2 | 3 | 5 |
| 2024 | 0 | 2 | 2 |
To return to the timeline,
click here.
Below are the most recent publications written about "Myocytes, Cardiac" by people in Profiles.
-
Blink nadir measurements of sarcoplasmic reticulum are consistent with strong local Ca2+ depletion. Biophys J. 2025 Jan 21; 124(2):245-255.
-
Alda-1 attenuation of binge alcohol-caused atrial arrhythmias through a novel mechanism of suppressed c-Jun N-terminal Kinase-2 activity. J Mol Cell Cardiol. 2024 12; 197:11-19.
-
Role of Mitochondrial ROS for Calcium Alternans in Atrial Myocytes. Biomolecules. 2024 Jan 24; 14(2).
-
The 'Reverse FDUF' Mechanism of Atrial Excitation-Contraction Coupling Sustains Calcium Alternans-A Hypothesis. Biomolecules. 2022 12 20; 13(1).
-
The HVCN1 voltage-gated proton channel contributes to pH regulation in canine ventricular myocytes. J Physiol. 2022 05; 600(9):2089-2103.
-
Inositol 1,4,5-trisphosphate receptor - reactive oxygen signaling domain regulates excitation-contraction coupling in atrial myocytes. J Mol Cell Cardiol. 2022 02; 163:147-155.
-
Impaired Binding to Junctophilin-2 and Nanostructural Alteration in CPVT Mutation. Circ Res. 2021 07 23; 129(3):e35-e52.
-
Excitation-contraction coupling and calcium release in atrial muscle. Pflugers Arch. 2021 03; 473(3):317-329.
-
Mitochondrial calcium uniporter complex activation protects against calcium alternans in atrial myocytes. Am J Physiol Heart Circ Physiol. 2020 10 01; 319(4):H873-H881.
-
Amorphous SiO2 nanoparticles promote cardiac dysfunction via the opening of the mitochondrial permeability transition pore in rat heart and human cardiomyocytes. Part Fibre Toxicol. 2020 May 07; 17(1):15.