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Giedrius Kanaporis

TitleAssistant Professor
InstitutionRush University, Rush Medical College
DepartmentPhysiology and Biophysics
Address1750 West Harrison St
Chicago IL 60612
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    Collapse Overview 
    Collapse overview
    My Scopus ID is 21742760800.
    My NIH COMMONS name is GKANAPORIS.

    Research Areas:
    Cardiac electrophysiology, cardiac arrhythmias, heart failure, intracellular calcium signaling in subcellular domains, cell-to-cell coupling.

    My Faculty Profile at Rush University Medical Center:
    https://www.rushu.rush.edu/faculty/giedrius-kanaporis-phd

    My Laboratory:
    https://www.rushu.rush.edu/research/departmental-research/physiology-and-biophysics-research/laboratory-lothar-blatter-md-dr-med

    My NCBI Bibliography:
    https://www.ncbi.nlm.nih.gov/myncbi/browse/collection/48071495/?sort=date&direction=descending

    My Scopus:
    https://www.scopus.com/authid/detail.uri?authorId=21742760800

    Education:
    PhD, Kaunas University of Medicine, Kaunas, Lithuania
    MS, Vytautas Magnus University, Kaunas, Lithuania
    BS, Vytautas Magnus University, Kaunas, Lithuania

    Collapse Bibliographic 
    Collapse selected publications
    Publications listed below are automatically derived from MEDLINE/PubMed and other sources, which might result in incorrect or missing publications. Faculty can login to make corrections and additions.
    Newest   |   Oldest   |   Most Cited   |   Most Discussed   |   Timeline   |   Field Summary   |   Plain Text
    PMC Citations indicate the number of times the publication was cited by articles in PubMed Central, and the Altmetric score represents citations in news articles and social media. (Note that publications are often cited in additional ways that are not shown here.) Fields are based on how the National Library of Medicine (NLM) classifies the publication's journal and might not represent the specific topic of the publication. Translation tags are based on the publication type and the MeSH terms NLM assigns to the publication. Some publications (especially newer ones and publications not in PubMed) might not yet be assigned Field or Translation tags.) Click a Field or Translation tag to filter the publications.
    1. Kanaporis G, Blatter LA. Increased Risk for Atrial Alternans in Rabbit Heart Failure: The Role of Ca2+/Calmodulin-Dependent Kinase II and Inositol-1,4,5-trisphosphate Signaling. Biomolecules. 2023 Dec 30; 14(1). PMID: 38254653; PMCID: PMC10813785.
      Citations:    
    2. Kanaporis G, Martinez-Hernandez E, Blatter LA. Calcium- and voltage-driven atrial alternans: Insight from [Ca]i and Vm asynchrony. Physiol Rep. 2023 05; 11(10):e15703. PMID: 37226365.
      Citations:    
    3. Kanaporis G, Blatter LA. Activation of small conductance Ca2+ -activated K+ channels suppresses Ca2+ transient and action potential alternans in ventricular myocytes. J Physiol. 2023 01; 601(1):51-67. PMID: 36426548.
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    4. Martinez-Hernandez E, Kanaporis G, Blatter LA. Mechanism of carvedilol induced action potential and calcium alternans. Channels (Austin). 2022 12; 16(1):97-112. PMID: 35501948.
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    5. Martinez-Hernandez E, Blatter LA, Kanaporis G. L-type Ca2+ channel recovery from inactivation in rabbit atrial myocytes. Physiol Rep. 2022 03; 10(5):e15222. PMID: 35274829.
      Citations:    
    6. Blatter LA, Kanaporis G, Martinez-Hernandez E, Oropeza-Almazan Y, Banach K, Blatter LA, Kanaporis G, Martinez-Hernandez E, Oropeza-Almazan Y, Banach K. Excitation-contraction coupling and calcium release in atrial muscle. Pflugers Arch. 2021 03; 473(3):317-329. PMID: 33398498; PMCID: PMC7940565.
      Citations:    
    7. Gussak G, Marszalec W, Yoo S, Modi R, O'Callaghan C, Aistrup GL, Cordeiro JM, Goodrow R, Kanaporis G, Blatter LA, Shiferaw Y, Arora R, Zhou J, Burrell AR, Wasserstrom JA. Triggered Ca2+ Waves Induce Depolarization of Maximum Diastolic Potential and Action Potential Prolongation in Dog Atrial Myocytes. Circ Arrhythm Electrophysiol. 2020 06; 13(6):e008179. PMID: 32433891.
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    8. Zhu K, Lai Y, Cao H, Bai X, Liu C, Yan Q, Ma L, Chen D, Kanaporis G, Wang J, Li L, Cheng T, Wang Y, Wu C, Xiao G. Kindlin-2 modulates MafA and ?-catenin expression to regulate ?-cell function and mass in mice. Nat Commun. 2020 01 24; 11(1):484. PMID: 31980627.
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    9. Treinys R, Kanaporis G, Fischmeister R, Jurevicius J. Metabolic Inhibition Induces Transient Increase of L-type Ca2+ Current in Human and Rat Cardiac Myocytes. Int J Mol Sci. 2019 Mar 26; 20(6). PMID: 30917498.
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    10. Kanaporis G, Kalik ZM, Blatter LA. Action potential shortening rescues atrial calcium alternans. J Physiol. 2019 02; 597(3):723-740. PMID: 30412286.
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    11. Kanaporis G, Treinys R, Fischmeister R, Jurevicius J. Metabolic inhibition reduces cardiac L-type Ca2+ channel current due to acidification caused by ATP hydrolysis. PLoS One. 2017; 12(8):e0184246. PMID: 28859158.
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    12. Kanaporis G, Blatter LA. Alternans in atria: Mechanisms and clinical relevance. Medicina (Kaunas). 2017; 53(3):139-149. PMID: 28666575.
      Citations:    
    13. Kanaporis G, Blatter LA. AP and Ca2+ alternans: An inseparable couple. Channels (Austin). 2017 09 03; 11(5):368-369. PMID: 28498780.
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    14. Kanaporis G, Blatter LA. Membrane potential determines calcium alternans through modulation of SR Ca2+ load and L-type Ca2+ current. J Mol Cell Cardiol. 2017 04; 105:49-58. PMID: 28257761.
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    15. Kanaporis G, Blatter LA. Ca(2+)-activated chloride channel activity during Ca(2+) alternans in ventricular myocytes. Channels (Austin). 2016 Nov; 10(6):507-17. PMID: 27356267.
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    16. Kanaporis G, Blatter LA. Calcium-activated chloride current determines action potential morphology during calcium alternans in atrial myocytes. J Physiol. 2016 Feb 01; 594(3):699-714. PMID: 26662365.
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    17. Kanaporis G, Blatter LA. The mechanisms of calcium cycling and action potential dynamics in cardiac alternans. Circ Res. 2015 Feb 27; 116(5):846-56. PMID: 25532796.
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    18. Kanaporis G, Marti?iene I, Jurevicius J, Vosyliute R, Navalinskas A, Treinys R, Matiukas A, Pertsov AM. Optical mapping at increased illumination intensities. J Biomed Opt. 2012 Sep; 17(9):96007-1. PMID: 23085908.
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    19. Magome N, Kanaporis G, Moisan N, Tanaka K, Agladze K. Photo-control of excitation waves in cardiomyocyte tissue culture. Tissue Eng Part A. 2011 Nov; 17(21-22):2703-11. PMID: 21834666.
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    20. Kanaporis G, Brink PR, Valiunas V. Gap junction permeability: selectivity for anionic and cationic probes. Am J Physiol Cell Physiol. 2011 Mar; 300(3):C600-9. PMID: 21148413.
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    21. Valiunas V, Kanaporis G, Valiuniene L, Gordon C, Wang HZ, Li L, Robinson RB, Rosen MR, Cohen IS, Brink PR. Coupling an HCN2-expressing cell to a myocyte creates a two-cell pacing unit. J Physiol. 2009 Nov 01; 587(Pt 21):5211-26. PMID: 19736302.
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    22. Kanaporis G, Mese G, Valiuniene L, White TW, Brink PR, Valiunas V. Gap junction channels exhibit connexin-specific permeability to cyclic nucleotides. J Gen Physiol. 2008 Apr; 131(4):293-305. PMID: 18378798.
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    23. Thomas BC, Minogue PJ, Valiunas V, Kanaporis G, Brink PR, Berthoud VM, Beyer EC. Cataracts are caused by alterations of a critical N-terminal positive charge in connexin50. Invest Ophthalmol Vis Sci. 2008 Jun; 49(6):2549-56. PMID: 18326694.
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    24. Yum SW, Zhang J, Valiunas V, Kanaporis G, Brink PR, White TW, Scherer SS. Human connexin26 and connexin30 form functional heteromeric and heterotypic channels. Am J Physiol Cell Physiol. 2007 Sep; 293(3):C1032-48. PMID: 17615163.
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    25. Zablockaite D, Gendviliene V, Macianskiene R, Skeberdis VA, Jurevicius J, Kanaporis G, Gurskaite H, Benetis R. Effect of hyperosmolarity on beta2-adrenergic stimulation in human atrium. Medicina (Kaunas). 2005; 41(5):401-8. PMID: 15947524.
      Citations:    
    26. Zablockaite D, Gendviliene V, Macianskiene R, Skeberdis VA, Jurevicius J, Kanaporis G, Benetis R. Changes of beta2-adrenergic stimulation induced by hyperosmosis in human atrium. Rocz Akad Med Bialymst. 2005; 50:244-6. PMID: 16358976.
      Citations:    
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