"Myocytes, Cardiac" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Striated muscle cells found in the heart. They are derived from cardiac myoblasts (MYOBLASTS, CARDIAC).
| Descriptor ID |
D032383
|
| MeSH Number(s) |
A07.541.704.570 A10.690.552.750.570 A11.620.500
|
| Concept/Terms |
Myocytes, Cardiac- Myocytes, Cardiac
- Cardiac Myocyte
- Cardiac Myocytes
- Myocyte, Cardiac
- Muscle Cells, Heart
- Cell, Heart Muscle
- Cells, Heart Muscle
- Heart Muscle Cell
- Heart Muscle Cells
- Muscle Cell, Heart
- Cardiomyocytes
- Cardiomyocyte
- Muscle Cells, Cardiac
- Cardiac Muscle Cell
- Cardiac Muscle Cells
- Cell, Cardiac Muscle
- Cells, Cardiac Muscle
- Muscle Cell, Cardiac
|
Below are MeSH descriptors whose meaning is more general than "Myocytes, Cardiac".
Below are MeSH descriptors whose meaning is more specific than "Myocytes, Cardiac".
This graph shows the total number of publications written about "Myocytes, Cardiac" by people in this website by year, and whether "Myocytes, Cardiac" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2002 | 2 | 0 | 2 |
| 2003 | 8 | 0 | 8 |
| 2004 | 6 | 2 | 8 |
| 2005 | 5 | 4 | 9 |
| 2006 | 7 | 0 | 7 |
| 2007 | 9 | 1 | 10 |
| 2008 | 7 | 1 | 8 |
| 2009 | 6 | 0 | 6 |
| 2010 | 6 | 1 | 7 |
| 2011 | 6 | 2 | 8 |
| 2012 | 8 | 2 | 10 |
| 2013 | 9 | 2 | 11 |
| 2014 | 8 | 0 | 8 |
| 2015 | 2 | 0 | 2 |
| 2016 | 4 | 0 | 4 |
| 2017 | 6 | 2 | 8 |
| 2018 | 3 | 1 | 4 |
| 2019 | 2 | 0 | 2 |
| 2020 | 3 | 0 | 3 |
| 2021 | 2 | 2 | 4 |
| 2022 | 2 | 3 | 5 |
| 2024 | 0 | 1 | 1 |
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click here.
Below are the most recent publications written about "Myocytes, Cardiac" by people in Profiles.
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Role of Mitochondrial ROS for Calcium Alternans in Atrial Myocytes. Biomolecules. 2024 Jan 24; 14(2).
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Long-Term Alcohol-Activated c-Jun N-terminal Kinase Isoform 2 Preserves Cardiac Function but Drives Ca2+-Triggered Arrhythmias. Cells. 2023 09 08; 12(18).
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Renal Denervation Helps Preserve the Ejection Fraction by Preserving Endocardial-Endothelial Function during Heart Failure. Int J Mol Sci. 2023 Apr 15; 24(8).
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Ero1a-Dependent ERp44 Dissociation From RyR2 Contributes to Cardiac Arrhythmia. Circ Res. 2022 03 04; 130(5):711-724.
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Inositol 1,4,5-trisphosphate receptor - reactive oxygen signaling domain regulates excitation-contraction coupling in atrial myocytes. J Mol Cell Cardiol. 2022 02; 163:147-155.
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Upregulation of transient receptor potential melastatin 4 (TRPM4) in ventricular fibroblasts from heart failure patients. Pflugers Arch. 2021 03; 473(3):521-531.
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Stress-driven cardiac calcium mishandling via a kinase-to-kinase crosstalk. Pflugers Arch. 2021 03; 473(3):363-375.
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Excitation-contraction coupling and calcium release in atrial muscle. Pflugers Arch. 2021 03; 473(3):317-329.
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JNK2, a Newly-Identified SERCA2 Enhancer, Augments an Arrhythmic [Ca2+]SR Leak-Load Relationship. Circ Res. 2021 02 19; 128(4):455-470.
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Secreted Protein Acidic and Cysteine Rich Matricellular Protein is Enriched in the Bioactive Fraction of the Human Vascular Pericyte Secretome. Antioxid Redox Signal. 2021 05 20; 34(15):1151-1164.