 Myocytes, Cardiac
"Myocytes, Cardiac" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Striated muscle cells found in the heart. They are derived from cardiac myoblasts (MYOBLASTS, CARDIAC).
| Descriptor ID |
D032383
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| MeSH Number(s) |
A07.541.704.570 A10.690.552.750.570 A11.620.500
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| Concept/Terms |
Myocytes, Cardiac- Myocytes, Cardiac
- Cardiac Myocyte
- Cardiac Myocytes
- Myocyte, Cardiac
- Cardiomyocytes
- Cardiomyocyte
- Muscle Cells, Cardiac
- Cardiac Muscle Cell
- Cardiac Muscle Cells
- Cell, Cardiac Muscle
- Cells, Cardiac Muscle
- Muscle Cell, Cardiac
- Muscle Cells, Heart
- Cell, Heart Muscle
- Cells, Heart Muscle
- Heart Muscle Cell
- Heart Muscle Cells
- Muscle Cell, Heart
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Below are MeSH descriptors whose meaning is more general than "Myocytes, Cardiac".
Below are MeSH descriptors whose meaning is more specific than "Myocytes, Cardiac".
This graph shows the total number of publications written about "Myocytes, Cardiac" by people in this website by year, and whether "Myocytes, Cardiac" was a major or minor topic of these publications.
To see the data from this visualization as text, click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2002 | 2 | 1 | 3 | | 2003 | 9 | 0 | 9 | | 2004 | 6 | 4 | 10 | | 2005 | 5 | 6 | 11 | | 2006 | 8 | 1 | 9 | | 2007 | 9 | 1 | 10 | | 2008 | 10 | 1 | 11 | | 2009 | 7 | 3 | 10 | | 2010 | 6 | 2 | 8 | | 2011 | 6 | 2 | 8 | | 2012 | 9 | 2 | 11 | | 2013 | 10 | 2 | 12 | | 2014 | 9 | 1 | 10 | | 2015 | 3 | 2 | 5 | | 2016 | 4 | 0 | 4 | | 2017 | 5 | 2 | 7 | | 2018 | 1 | 0 | 1 |
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Below are the most recent publications written about "Myocytes, Cardiac" by people in Profiles.
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DeSantiago J, Bare DJ, Varma D, Solaro RJ, Arora R, Banach K. Loss of p21-activated kinase 1 (Pak1) promotes atrial arrhythmic activity. Heart Rhythm. 2018 08; 15(8):1233-1241.
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Kanaporis G, Treinys R, Fischmeister R, Jurevicius J. Metabolic inhibition reduces cardiac L-type Ca2+ channel current due to acidification caused by ATP hydrolysis. PLoS One. 2017; 12(8):e0184246.
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Pereira L, Bare DJ, Galice S, Shannon TR, Bers DM. ß-Adrenergic induced SR Ca2+ leak is mediated by an Epac-NOS pathway. J Mol Cell Cardiol. 2017 07; 108:8-16.
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Aguilar-Sanchez Y, Fainstein D, Mejia-Alvarez R, Escobar AL. Local Field Fluorescence Microscopy: Imaging Cellular Signals in Intact Hearts. J Vis Exp. 2017 03 08; (121).
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Kanaporis G, Blatter LA. Membrane potential determines calcium alternans through modulation of SR Ca2+ load and L-type Ca2+ current. J Mol Cell Cardiol. 2017 04; 105:49-58.
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Maxwell JT, Blatter LA. A novel mechanism of tandem activation of ryanodine receptors by cytosolic and SR luminal Ca2+ during excitation-contraction coupling in atrial myocytes. J Physiol. 2017 06 15; 595(12):3835-3845.
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Bovo E, Huke S, Blatter LA, Zima AV. The effect of PKA-mediated phosphorylation of ryanodine receptor on SR Ca2+ leak in ventricular myocytes. J Mol Cell Cardiol. 2017 03; 104:9-16.
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Uehara A, Murayama T, Yasukochi M, Fill M, Horie M, Okamoto T, Matsuura Y, Uehara K, Fujimoto T, Sakurai T, Kurebayashi N. Extensive Ca2+ leak through K4750Q cardiac ryanodine receptors caused by cytosolic and luminal Ca2+ hypersensitivity. J Gen Physiol. 2017 Feb; 149(2):199-218.
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Hohendanner F, DeSantiago J, Heinzel FR, Blatter LA. Dyssynchronous calcium removal in heart failure-induced atrial remodeling. Am J Physiol Heart Circ Physiol. 2016 12 01; 311(6):H1352-H1359.
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Hegyi B, Bányász T, Shannon TR, Chen-Izu Y, Izu LT. Electrophysiological Determination of Submembrane Na(+) Concentration in Cardiac Myocytes. Biophys J. 2016 Sep 20; 111(6):1304-1315.
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