Cyclic AMP Response Element-Binding Protein
"Cyclic AMP Response Element-Binding Protein" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A protein that has been shown to function as a calcium-regulated transcription factor as well as a substrate for depolarization-activated CALCIUM-CALMODULIN-DEPENDENT PROTEIN KINASES. This protein functions to integrate both calcium and cAMP signals.
| Descriptor ID |
D017362
|
| MeSH Number(s) |
D12.776.260.108.184 D12.776.930.127.184
|
| Concept/Terms |
Cyclic AMP Response Element-Binding Protein- Cyclic AMP Response Element-Binding Protein
- CREB Protein
- DNA-Binding Protein, Cyclic AMP-Responsive
- DNA Binding Protein, Cyclic AMP Responsive
- cAMP Response Element-Binding Protein
- cAMP Response Element Binding Protein
- Cyclic AMP-Responsive DNA-Binding Protein
- Cyclic AMP Responsive DNA Binding Protein
|
Below are MeSH descriptors whose meaning is more general than "Cyclic AMP Response Element-Binding Protein".
Below are MeSH descriptors whose meaning is more specific than "Cyclic AMP Response Element-Binding Protein".
This graph shows the total number of publications written about "Cyclic AMP Response Element-Binding Protein" by people in this website by year, and whether "Cyclic AMP Response Element-Binding Protein" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2003 | 0 | 1 | 1 |
| 2006 | 2 | 0 | 2 |
| 2007 | 0 | 1 | 1 |
| 2008 | 0 | 1 | 1 |
| 2012 | 0 | 1 | 1 |
| 2013 | 2 | 1 | 3 |
| 2014 | 0 | 1 | 1 |
| 2015 | 0 | 2 | 2 |
| 2016 | 0 | 1 | 1 |
| 2017 | 1 | 2 | 3 |
| 2018 | 0 | 3 | 3 |
| 2019 | 0 | 2 | 2 |
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Below are the most recent publications written about "Cyclic AMP Response Element-Binding Protein" by people in Profiles.
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Mode of Action of Aspirin in Experimental Autoimmune Encephalomyelitis. DNA Cell Biol. 2019 Jul; 38(7):593-596.
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Cinnamon and its Metabolite Protect the Nigrostriatum in a Mouse Model of Parkinson's Disease Via Astrocytic GDNF. J Neuroimmune Pharmacol. 2019 09; 14(3):503-518.
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Aspirin ameliorates experimental autoimmune encephalomyelitis through interleukin-11-mediated protection of regulatory T cells. Sci Signal. 2018 11 27; 11(558).
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Low-Dose Aspirin Upregulates Tyrosine Hydroxylase and Increases Dopamine Production in Dopaminergic Neurons: Implications for Parkinson's Disease. J Neuroimmune Pharmacol. 2019 06; 14(2):173-187.
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Aspirin binds to PPARa to stimulate hippocampal plasticity and protect memory. Proc Natl Acad Sci U S A. 2018 07 31; 115(31):E7408-E7417.
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Upregulation of Myelin Gene Expression by a Physically-Modified Saline via Phosphatidylinositol 3-Kinase-Mediated Activation of CREB: Implications for Multiple Sclerosis. Neurochem Res. 2018 Feb; 43(2):407-419.
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CREB Protein Mediates Alcohol-Induced Circadian Disruption and Intestinal Permeability. Alcohol Clin Exp Res. 2017 Dec; 41(12):2007-2014.
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Selective lowering of synapsins induced by oligomeric a-synuclein exacerbates memory deficits. Proc Natl Acad Sci U S A. 2017 06 06; 114(23):E4648-E4657.
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Cinnamon Converts Poor Learning Mice to Good Learners: Implications for Memory Improvement. J Neuroimmune Pharmacol. 2016 12; 11(4):693-707.
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Sodium Benzoate, a Metabolite of Cinnamon and a Food Additive, Upregulates Ciliary Neurotrophic Factor in Astrocytes and Oligodendrocytes. Neurochem Res. 2015 Nov; 40(11):2333-47.