"Insulin-Secreting Cells" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A type of pancreatic cell representing about 50-80% of the islet cells. Beta cells secrete INSULIN.
Descriptor ID |
D050417
|
MeSH Number(s) |
A03.734.414.131 A06.390.131 A06.407.414.087 A11.382.625.092 A11.436.294.092
|
Concept/Terms |
Insulin-Secreting Cells- Insulin-Secreting Cells
- Cell, Insulin-Secreting
- Cells, Insulin-Secreting
- Insulin Secreting Cells
- Insulin-Secreting Cell
- beta Cells, Pancreatic
- Pancreatic beta Cell
- beta Cell, Pancreatic
- Pancreatic B Cells
- B Cell, Pancreatic
- B Cells, Pancreatic
- Pancreatic B Cell
- Pancreatic beta Cells
|
Below are MeSH descriptors whose meaning is more general than "Insulin-Secreting Cells".
Below are MeSH descriptors whose meaning is more specific than "Insulin-Secreting Cells".
This graph shows the total number of publications written about "Insulin-Secreting Cells" by people in this website by year, and whether "Insulin-Secreting Cells" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
Year | Major Topic | Minor Topic | Total |
---|
2009 | 0 | 2 | 2 |
2011 | 1 | 0 | 1 |
2014 | 1 | 0 | 1 |
2015 | 2 | 0 | 2 |
2016 | 1 | 0 | 1 |
2017 | 0 | 1 | 1 |
2018 | 1 | 0 | 1 |
2020 | 2 | 0 | 2 |
To return to the timeline,
click here.
Below are the most recent publications written about "Insulin-Secreting Cells" by people in Profiles.
-
Markers of acute toxicity of DDT exposure in pancreatic beta-cells determined by a proteomic approach. PLoS One. 2020; 15(10):e0229430.
-
Effect of short-term prednisone on beta-cell function in subjects with type 2 diabetes mellitus and healthy subjects. PLoS One. 2020; 15(5):e0231190.
-
Kindlin-2 modulates MafA and ?-catenin expression to regulate ?-cell function and mass in mice. Nat Commun. 2020 01 24; 11(1):484.
-
HDAC1 overexpression enhances ?-cell proliferation by down-regulating Cdkn1b/p27. Biochem J. 2018 12 19; 475(24):3997-4010.
-
Distinguishing signal from autofluorescence in cryogenic correlated light and electron microscopy of mammalian cells. J Struct Biol. 2018 01; 201(1):15-25.
-
?-Cell deletion of Nr4a1 and Nr4a3 nuclear receptors impedes mitochondrial respiration and insulin secretion. Am J Physiol Endocrinol Metab. 2016 07 01; 311(1):E186-201.
-
Cdk5r1 Overexpression Induces Primary ?-Cell Proliferation. J Diabetes Res. 2016; 2016:6375804.
-
Aurora Kinase A is critical for the Nkx6.1 mediated ?-cell proliferation pathway. Islets. 2015; 7(1):e1027854.
-
Maternal inheritance of an inactive type III deiodinase gene allele affects mouse pancreatic ?-cells and disrupts glucose homeostasis. Endocrinology. 2014 Aug; 155(8):3160-71.
-
The thyroid hormone-inactivating type III deiodinase is expressed in mouse and human beta-cells and its targeted inactivation impairs insulin secretion. Endocrinology. 2011 Oct; 152(10):3717-27.