"Apoptosis" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
One of the mechanisms by which CELL DEATH occurs (compare with NECROSIS and AUTOPHAGOCYTOSIS). Apoptosis is the mechanism responsible for the physiological deletion of cells and appears to be intrinsically programmed. It is characterized by distinctive morphologic changes in the nucleus and cytoplasm, chromatin cleavage at regularly spaced sites, and the endonucleolytic cleavage of genomic DNA; (DNA FRAGMENTATION); at internucleosomal sites. This mode of cell death serves as a balance to mitosis in regulating the size of animal tissues and in mediating pathologic processes associated with tumor growth.
Descriptor ID |
D017209
|
MeSH Number(s) |
G04.146.160
|
Concept/Terms |
Apoptosis- Apoptosis
- Programmed Cell Death, Type I
|
Below are MeSH descriptors whose meaning is more general than "Apoptosis".
Below are MeSH descriptors whose meaning is more specific than "Apoptosis".
This graph shows the total number of publications written about "Apoptosis" by people in this website by year, and whether "Apoptosis" was a major or minor topic of these publications.
To see the data from this visualization as text,
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Year | Major Topic | Minor Topic | Total |
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1994 | 1 | 0 | 1 |
1995 | 1 | 0 | 1 |
1996 | 5 | 2 | 7 |
1997 | 1 | 4 | 5 |
1998 | 2 | 2 | 4 |
1999 | 6 | 3 | 9 |
2000 | 2 | 1 | 3 |
2001 | 4 | 2 | 6 |
2002 | 5 | 4 | 9 |
2003 | 6 | 5 | 11 |
2004 | 6 | 7 | 13 |
2005 | 2 | 8 | 10 |
2006 | 2 | 2 | 4 |
2007 | 9 | 4 | 13 |
2008 | 2 | 4 | 6 |
2009 | 4 | 2 | 6 |
2010 | 1 | 4 | 5 |
2011 | 1 | 5 | 6 |
2012 | 2 | 5 | 7 |
2013 | 4 | 6 | 10 |
2014 | 3 | 8 | 11 |
2015 | 1 | 4 | 5 |
2016 | 0 | 3 | 3 |
2017 | 2 | 9 | 11 |
2018 | 1 | 8 | 9 |
2019 | 2 | 3 | 5 |
2020 | 0 | 1 | 1 |
2021 | 1 | 1 | 2 |
2022 | 0 | 4 | 4 |
2023 | 0 | 1 | 1 |
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Below are the most recent publications written about "Apoptosis" by people in Profiles.
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cIAP1/2 Antagonism Induces Antigen-Specific T Cell-Dependent Immunity. J Immunol. 2023 04 01; 210(7):991-1003.
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Lymphatic cells do not functionally integrate into 3D organotypic brain slice cultures, but aggregate around penetrating blood vessels. Exp Brain Res. 2022 Sep; 240(9):2349-2358.
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Wnt/?-Catenin Protects Lymphocytes from HIV-Mediated Apoptosis via Induction of Bcl-xL. Viruses. 2022 07 02; 14(7).
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Canady Cold Helios Plasma Reduces Soft Tissue Sarcoma Viability by Inhibiting Proliferation, Disrupting Cell Cycle, and Inducing Apoptosis: A Preliminary Report. Molecules. 2022 Jun 29; 27(13).
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Presence of CrkI-containing microvesicles in squamous cell carcinomas could have ramifications on tumor biology and cancer therapeutics. Sci Rep. 2022 03 21; 12(1):4803.
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Xenogeneic fibroblasts inhibit the growth of the breast and ovarian cancer cell lines in co-culture. Neoplasma. 2021 Nov; 68(6):1265-1271.
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Punctate Anetoderma After Colony-Stimulating Factor 1 Receptor and Programmed Cell Death 1 Blockade With Irradiation: Clinicopathologic Characterization of a Novel Eruption. JAMA Dermatol. 2021 08 01; 157(8):998-1000.
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Mitochondrial Functions Are Compromised in CD4 T Cells From ART-Controlled PLHIV. Front Immunol. 2021; 12:658420.
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Characterization of Dopamine Receptor Associated Drugs on the Proliferation and Apoptosis of Prostate Cancer Cell Lines. Anticancer Agents Med Chem. 2021; 21(9):1160-1171.
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Telomere and ATM Dynamics in CD4 T-Cell Depletion in Active and Virus-Suppressed HIV Infections. J Virol. 2020 10 27; 94(22).