Adaptor Proteins, Vesicular Transport
"Adaptor Proteins, Vesicular Transport" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A class of proteins involved in the transport of molecules via TRANSPORT VESICLES. They perform functions such as binding to the cell membrane, capturing cargo molecules and promoting the assembly of CLATHRIN. The majority of adaptor proteins exist as multi-subunit complexes, however monomeric varieties have also been found.
| Descriptor ID |
D033942
|
| MeSH Number(s) |
D12.776.543.990.150
|
| Concept/Terms |
Clathrin Adaptors- Clathrin Adaptors
- Adaptors, Clathrin
- Clathrin-Associated Adaptors
- Adaptors, Clathrin-Associated
- Clathrin Associated Adaptors
- Clathrin-Associated Proteins
- Clathrin Associated Proteins
- Clathrin Assembly Proteins
- Assembly Proteins, Clathrin
|
Below are MeSH descriptors whose meaning is more general than "Adaptor Proteins, Vesicular Transport".
Below are MeSH descriptors whose meaning is more specific than "Adaptor Proteins, Vesicular Transport".
This graph shows the total number of publications written about "Adaptor Proteins, Vesicular Transport" by people in this website by year, and whether "Adaptor Proteins, Vesicular Transport" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2003 | 1 | 0 | 1 |
| 2004 | 0 | 1 | 1 |
| 2007 | 1 | 0 | 1 |
| 2009 | 1 | 1 | 2 |
| 2011 | 2 | 0 | 2 |
| 2012 | 0 | 1 | 1 |
| 2013 | 2 | 1 | 3 |
| 2015 | 1 | 0 | 1 |
| 2016 | 2 | 0 | 2 |
| 2017 | 2 | 0 | 2 |
| 2020 | 0 | 1 | 1 |
| 2021 | 0 | 1 | 1 |
To return to the timeline,
click here.
Below are the most recent publications written about "Adaptor Proteins, Vesicular Transport" by people in Profiles.
-
Microglial Correlates of Late Life Physical Activity: Relationship with Synaptic and Cognitive Aging in Older Adults. J Neurosci. 2022 01 12; 42(2):288-298.
-
Cortical Proteins Associated With Cognitive Resilience in Community-Dwelling Older Persons. JAMA Psychiatry. 2020 11 01; 77(11):1172-1180.
-
Intersectin-1s deficiency in pulmonary pathogenesis. Respir Res. 2017 09 06; 18(1):168.
-
Modulation of Intersectin-1s Lung Expression Induces Obliterative Remodeling and Severe Plexiform Arteriopathy in the Murine Pulmonary Vascular Bed. Am J Pathol. 2017 Mar; 187(3):528-542.
-
Presynaptic proteins complexin-I and complexin-II differentially influence cognitive function in early and late stages of Alzheimer's disease. Acta Neuropathol. 2017 03; 133(3):395-407.
-
Rac1-mediated cytoskeleton rearrangements induced by intersectin-1s deficiency promotes lung cancer cell proliferation, migration and metastasis. Mol Cancer. 2016 09 14; 15(1):59.
-
Endocytic deficiency induced by ITSN-1s knockdown alters the Smad2/3-Erk1/2 signaling balance downstream of Alk5. J Cell Sci. 2015 Apr 15; 128(8):1528-41.
-
A novel p38 mitogen-activated protein kinase/Elk-1 transcription factor-dependent molecular mechanism underlying abnormal endothelial cell proliferation in plexogenic pulmonary arterial hypertension. J Biol Chem. 2013 Sep 06; 288(36):25701-25716.
-
Long-term silencing of intersectin-1s in mouse lungs by repeated delivery of a specific siRNA via cationic liposomes. Evaluation of knockdown effects by electron microscopy. J Vis Exp. 2013 Jun 21; (76).
-
In vivo knockdown of intersectin-1s alters endothelial cell phenotype and causes microvascular remodeling in the mouse lungs. Apoptosis. 2013 Jan; 18(1):57-76.