"Long-Term Potentiation" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A persistent increase in synaptic efficacy, usually induced by appropriate activation of the same synapses. The phenomenological properties of long-term potentiation suggest that it may be a cellular mechanism of learning and memory.
| Descriptor ID |
D017774
|
| MeSH Number(s) |
G11.561.638.350
|
| Concept/Terms |
Long-Term Potentiation- Long-Term Potentiation
- Long Term Potentiation
- Long-Term Potentiations
- Potentiation, Long-Term
- Potentiations, Long-Term
|
Below are MeSH descriptors whose meaning is more general than "Long-Term Potentiation".
Below are MeSH descriptors whose meaning is more specific than "Long-Term Potentiation".
This graph shows the total number of publications written about "Long-Term Potentiation" by people in this website by year, and whether "Long-Term Potentiation" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2003 | 1 | 0 | 1 |
| 2006 | 2 | 1 | 3 |
| 2010 | 1 | 0 | 1 |
| 2011 | 1 | 1 | 2 |
| 2013 | 0 | 1 | 1 |
| 2015 | 1 | 1 | 2 |
| 2020 | 0 | 1 | 1 |
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Below are the most recent publications written about "Long-Term Potentiation" by people in Profiles.
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Limiting RyR2 Open Time Prevents Alzheimer's Disease-Related Neuronal Hyperactivity and Memory Loss but Not ?-Amyloid Accumulation. Cell Rep. 2020 09 22; 32(12):108169.
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Fear potentiated startle increases phospholipase D (PLD) expression/activity and PLD-linked metabotropic glutamate receptor mediated post-tetanic potentiation in rat amygdala. Neurobiol Learn Mem. 2016 Feb; 128:65-79.
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B-lymphocyte-mediated delayed cognitive impairment following stroke. J Neurosci. 2015 Feb 04; 35(5):2133-45.
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Development of Novel In Vivo Chemical Probes to Address CNS Protein Kinase Involvement in Synaptic Dysfunction. PLoS One. 2013; 8(6):e66226.
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Dopamine-induced plasticity, phospholipase D (PLD) activity and cocaine-cue behavior depend on PLD-linked metabotropic glutamate receptors in amygdala. PLoS One. 2011; 6(9):e25639.
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Cocaine withdrawal reduces group I mGluR-mediated long-term potentiation via decreased GABAergic transmission in the amygdala. Eur J Neurosci. 2011 Jul; 34(2):177-89.
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Dopamine receptor mechanisms mediate corticotropin-releasing factor-induced long-term potentiation in the rat amygdala following cocaine withdrawal. Eur J Neurosci. 2010 Mar; 31(6):1027-42.
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Long-term potentiation (LTP) in the central amygdala (CeA) is enhanced after prolonged withdrawal from chronic cocaine and requires CRF1 receptors. J Neurophysiol. 2007 Jan; 97(1):937-41.
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Effect of CX516, an AMPA-modulating compound, on cognition and behavior in fragile X syndrome: a controlled trial. J Child Adolesc Psychopharmacol. 2006 Oct; 16(5):525-40.
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Cocaine withdrawal enhances long-term potentiation induced by corticotropin-releasing factor at central amygdala glutamatergic synapses via CRF, NMDA receptors and PKA. Eur J Neurosci. 2006 Sep; 24(6):1733-43.