Plaque, Amyloid
"Plaque, Amyloid" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Accumulations of extracellularly deposited AMYLOID FIBRILS within tissues.
Descriptor ID |
D058225
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MeSH Number(s) |
C23.300.821
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Concept/Terms |
Plaque, Amyloid- Plaque, Amyloid
- Amyloid Plaque
- Amyloid Plaques
- Plaques, Amyloid
- Amyloid Deposits
- Amyloid Deposit
- Deposit, Amyloid
- Deposits, Amyloid
Senile Plaques- Senile Plaques
- Plaque, Senile
- Plaques, Senile
- Senile Plaque
- Neuritic Plaques
- Neuritic Plaque
- Plaque, Neuritic
- Plaques, Neuritic
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Below are MeSH descriptors whose meaning is more general than "Plaque, Amyloid".
Below are MeSH descriptors whose meaning is more specific than "Plaque, Amyloid".
This graph shows the total number of publications written about "Plaque, Amyloid" by people in this website by year, and whether "Plaque, Amyloid" was a major or minor topic of these publications.
To see the data from this visualization as text, click here.
Year | Major Topic | Minor Topic | Total |
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2002 | 1 | 1 | 2 | 2003 | 1 | 2 | 3 | 2004 | 1 | 0 | 1 | 2005 | 1 | 2 | 3 | 2006 | 0 | 2 | 2 | 2007 | 0 | 4 | 4 | 2008 | 1 | 3 | 4 | 2009 | 0 | 1 | 1 | 2010 | 0 | 3 | 3 | 2011 | 2 | 2 | 4 | 2012 | 2 | 3 | 5 | 2013 | 3 | 3 | 6 | 2014 | 3 | 3 | 6 | 2015 | 2 | 1 | 3 | 2016 | 0 | 2 | 2 | 2017 | 1 | 4 | 5 | 2018 | 6 | 0 | 6 | 2019 | 2 | 2 | 4 | 2021 | 1 | 5 | 6 | 2022 | 0 | 2 | 2 | 2023 | 0 | 1 | 1 |
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Below are the most recent publications written about "Plaque, Amyloid" by people in Profiles.
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Rangasamy SB, Jana M, Dasarathi S, Kundu M, Pahan K. Treadmill workout activates PPARa in the hippocampus to upregulate ADAM10, decrease plaques and improve cognitive functions in 5XFAD mouse model of Alzheimer's disease. Brain Behav Immun. 2023 03; 109:204-218.
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Katsumata Y, Shade LM, Hohman TJ, Schneider JA, Bennett DA, Farfel JM. Multiple gene variants linked to Alzheimer's-type clinical dementia via GWAS are also associated with non-Alzheimer's neuropathologic entities. Neurobiol Dis. 2022 Nov; 174:105880.
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Rabin JS, Nichols E, La Joie R, Casaletto KB, Palta P, Dams-O'Connor K, Kumar RG, George KM, Satizabal CL, Schneider JA, Pa J, Brickman AM. Cerebral amyloid angiopathy interacts with neuritic amyloid plaques to promote tau and cognitive decline. Brain. 2022 08 27; 145(8):2823-2833.
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Musiek ES, Bennett DA. Aducanumab and the "post-amyloid" era of Alzheimer research? Neuron. 2021 10 06; 109(19):3045-3047.
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Jeong A, Cheng S, Zhong R, Bennett DA, Berg? MO, Li L. Protein farnesylation is upregulated in Alzheimer's human brains and neuron-specific suppression of farnesyltransferase mitigates pathogenic processes in Alzheimer's model mice. Acta Neuropathol Commun. 2021 07 27; 9(1):129.
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Russo ML, Molina-Campos E, Ybarra N, Rogalsky AE, Musial TF, Jimenez V, Haddad LG, Voskobiynyk Y, D'Souza GX, Carballo G, Neuman KM, Chetkovich DM, Oh MM, Disterhoft JF, Nicholson DA. Variability in sub-threshold signaling linked to Alzheimer's disease emerges with age and amyloid plaque deposition in mouse ventral CA1 pyramidal neurons. Neurobiol Aging. 2021 10; 106:207-222.
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Nackenoff AG, Hohman TJ, Neuner SM, Akers CS, Weitzel NC, Shostak A, Ferguson SM, Mobley B, Bennett DA, Schneider JA, Jefferson AL, Kaczorowski CC, Schrag MS. PLD3 is a neuronal lysosomal phospholipase D associated with ?-amyloid plaques and cognitive function in Alzheimer's disease. PLoS Genet. 2021 04; 17(4):e1009406.
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Mehta RI, Schneider JA. What is 'Alzheimer's disease'? The neuropathological heterogeneity of clinically defined Alzheimer's dementia. Curr Opin Neurol. 2021 04 01; 34(2):237-245.
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Walker JM, Richardson TE, Farrell K, Iida MA, Foong C, Shang P, Attems J, Ayalon G, Beach TG, Bigio EH, Budson A, Cairns NJ, Corrada M, Cortes E, Dickson DW, Fischer P, Flanagan ME, Franklin E, Gearing M, Glass J, Hansen LA, Haroutunian V, Hof PR, Honig L, Kawas C, Keene CD, Kofler J, Kovacs GG, Lee EB, Lutz MI, Mao Q, Masliah E, McKee AC, McMillan CT, Mesulam MM, Murray M, Nelson PT, Perrin R, Pham T, Poon W, Purohit DP, Rissman RA, Sakai K, Sano M, Schneider JA, Stein TD, Teich AF, Trojanowski JQ, Troncoso JC, Vonsattel JP, Weintraub S, Wolk DA, Woltjer RL, Yamada M, Yu L, White CL, Crary JF. Early Selective Vulnerability of the CA2 Hippocampal Subfield in Primary Age-Related Tauopathy. J Neuropathol Exp Neurol. 2021 01 20; 80(2):102-111.
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Patel D, Roy A, Pahan K. PPARa serves as a new receptor of aspirin for neuroprotection. J Neurosci Res. 2020 04; 98(4):626-631.
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