"Receptors, Cell Surface" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Cell surface proteins that bind signalling molecules external to the cell with high affinity and convert this extracellular event into one or more intracellular signals that alter the behavior of the target cell (From Alberts, Molecular Biology of the Cell, 2nd ed, pp693-5). Cell surface receptors, unlike enzymes, do not chemically alter their ligands.
| Descriptor ID |
D011956
|
| MeSH Number(s) |
D12.776.543.750
|
| Concept/Terms |
|
Below are MeSH descriptors whose meaning is more general than "Receptors, Cell Surface".
Below are MeSH descriptors whose meaning is more specific than "Receptors, Cell Surface".
- Receptors, Cell Surface
- Asialoglycoprotein Receptor
- Autoreceptors
- CD36 Antigens
- Cysteine Loop Ligand-Gated Ion Channel Receptors
- Endoglin
- Endothelial Protein C Receptor
- Folate Receptors, GPI-Anchored
- Junctional Adhesion Molecule A
- Methyl-Accepting Chemotaxis Proteins
- Netrin Receptors
- Neuropilins
- Nogo Receptors
- Patched Receptors
- Receptor for Advanced Glycation End Products
- Receptor Protein-Tyrosine Kinases
- Receptors, Adipokine
- Receptors, Artificial
- Receptors, Autocrine Motility Factor
- Receptors, Biogenic Amine
- Receptors, Collagen
- Receptors, Death Domain
- Receptors, G-Protein-Coupled
- Receptors, Guanylate Cyclase-Coupled
- Receptors, Immunologic
- Receptors, Lipoprotein
- Receptors, N-Acetylglucosamine
- Receptors, Neurotransmitter
- Receptors, Notch
- Receptors, Peptide
- Receptors, Phospholipase A2
- Receptors, Proteinase-Activated
- Receptors, Transferrin
- Receptors, Urokinase Plasminogen Activator
- Receptors, Virus
- Receptors, Wnt
- Zona Pellucida Glycoproteins
This graph shows the total number of publications written about "Receptors, Cell Surface" by people in this website by year, and whether "Receptors, Cell Surface" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2001 | 1 | 0 | 1 |
| 2003 | 3 | 0 | 3 |
| 2004 | 1 | 0 | 1 |
| 2005 | 3 | 2 | 5 |
| 2006 | 0 | 1 | 1 |
| 2007 | 0 | 3 | 3 |
| 2008 | 0 | 1 | 1 |
| 2009 | 1 | 0 | 1 |
| 2010 | 2 | 0 | 2 |
| 2012 | 0 | 1 | 1 |
| 2015 | 1 | 0 | 1 |
| 2017 | 1 | 0 | 1 |
| 2018 | 0 | 1 | 1 |
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Below are the most recent publications written about "Receptors, Cell Surface" by people in Profiles.
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Targeted brain proteomics uncover multiple pathways to Alzheimer's dementia. Ann Neurol. 2018 07; 84(1):78-88.
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Identification of genes associated with dissociation of cognitive performance and neuropathological burden: Multistep analysis of genetic, epigenetic, and transcriptional data. PLoS Med. 2017 04; 14(4):e1002287.
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Soluble CD163 But Not Soluble CD14 Is Associated With Cytomegalovirus Immunoglobulin G Antibody Levels in Virologically Suppressed HIV+ Individuals. J Acquir Immune Defic Syndr. 2015 Dec 15; 70(5):e171-4.
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Activation of the Sonic Hedgehog pathway in thyroid neoplasms and its potential role in tumor cell proliferation. Endocr Relat Cancer. 2012 Apr; 19(2):167-79.
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Degranulation of natural killer cells following interaction with HIV-1-infected cells is hindered by downmodulation of NTB-A by Vpu. Cell Host Microbe. 2010 Nov 18; 8(5):397-409.
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Expression of IL-4 receptor alpha on smooth muscle cells is not necessary for development of experimental allergic asthma. J Allergy Clin Immunol. 2010 Aug; 126(2):347-54.
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The macrophage mannose receptor induces IL-17 in response to Candida albicans. Cell Host Microbe. 2009 Apr 23; 5(4):329-40.
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Dendritic cell interaction with Candida albicans critically depends on N-linked mannan. J Biol Chem. 2008 Jul 18; 283(29):20590-9.
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The zebrafish fleer gene encodes an essential regulator of cilia tubulin polyglutamylation. Mol Biol Cell. 2007 Nov; 18(11):4353-64.
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Human T-cell leukemia virus type 1 (HTLV-1) p12I down-modulates ICAM-1 and -2 and reduces adherence of natural killer cells, thereby protecting HTLV-1-infected primary CD4+ T cells from autologous natural killer cell-mediated cytotoxicity despite the reduction of major histocompatibility complex class I molecules on infected cells. J Virol. 2007 Sep; 81(18):9707-17.