Myocardial Reperfusion Injury
"Myocardial Reperfusion Injury" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Damage to the MYOCARDIUM resulting from MYOCARDIAL REPERFUSION (restoration of blood flow to ischemic areas of the HEART.) Reperfusion takes place when there is spontaneous thrombolysis, THROMBOLYTIC THERAPY, collateral flow from other coronary vascular beds, or reversal of vasospasm.
| Descriptor ID |
D015428
|
| MeSH Number(s) |
C14.280.238.615 C14.280.647.625 C14.907.585.625 C14.907.725.600 C23.550.767.877.500
|
| Concept/Terms |
Myocardial Reperfusion Injury- Myocardial Reperfusion Injury
- Injuries, Myocardial Reperfusion
- Myocardial Reperfusion Injuries
- Reperfusion Injuries, Myocardial
- Myocardial Ischemic Reperfusion Injury
- Reperfusion Injury, Myocardial
- Injury, Myocardial Reperfusion
|
Below are MeSH descriptors whose meaning is more general than "Myocardial Reperfusion Injury".
Below are MeSH descriptors whose meaning is more specific than "Myocardial Reperfusion Injury".
This graph shows the total number of publications written about "Myocardial Reperfusion Injury" by people in this website by year, and whether "Myocardial Reperfusion Injury" was a major or minor topic of these publications.
To see the data from this visualization as text,
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| Year | Major Topic | Minor Topic | Total |
|---|
| 1995 | 2 | 1 | 3 |
| 2007 | 1 | 0 | 1 |
| 2011 | 1 | 0 | 1 |
| 2013 | 1 | 1 | 2 |
| 2015 | 1 | 0 | 1 |
| 2017 | 0 | 1 | 1 |
| 2019 | 2 | 0 | 2 |
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Below are the most recent publications written about "Myocardial Reperfusion Injury" by people in Profiles.
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Changes in the Stoichiometry of Uniplex Decrease Mitochondrial Calcium Overload and Contribute to Tolerance of Cardiac Ischemia/Reperfusion Injury in Hypothyroidism. Thyroid. 2019 Dec; 29(12):1755-1764.
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HDL-Targeted Therapies During Myocardial Infarction. Cardiovasc Drugs Ther. 2019 06; 33(3):371-381.
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Ablation of the calpain-targeted site in cardiac myosin binding protein-C is cardioprotective during ischemia-reperfusion injury. J Mol Cell Cardiol. 2019 04; 129:236-246.
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Small Interfering RNA Targeting Mitochondrial Calcium Uniporter Improves Cardiomyocyte Cell Viability in Hypoxia/Reoxygenation Injury by Reducing Calcium Overload. Oxid Med Cell Longev. 2017; 2017:5750897.
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Circulating MicroRNAs as new biomarkers of ischaemia/reperfusion injury during cardiac surgery. Cardiology. 2015; 130(4):234-6.
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Distinct mPTP activation mechanisms in ischaemia-reperfusion: contributions of Ca2+, ROS, pH, and inorganic polyphosphate. Cardiovasc Res. 2015 May 01; 106(2):237-48.
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Ca2+ Sparks and Ca2+ waves are the subcellular events underlying Ca2+ overload during ischemia and reperfusion in perfused intact hearts. J Mol Cell Cardiol. 2015 Feb; 79:69-78.
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Report from a consensus conference on primary graft dysfunction after cardiac transplantation. J Heart Lung Transplant. 2014 Apr; 33(4):327-40.
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p21-Activated kinase1 (Pak1) is a negative regulator of NADPH-oxidase 2 in ventricular myocytes. J Mol Cell Cardiol. 2014 Feb; 67:77-85.
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Ischemia/Reperfusion injury protection by mesenchymal stem cell derived antioxidant capacity. Stem Cells Dev. 2013 Sep 15; 22(18):2497-507.