"Reperfusion Injury" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Adverse functional, metabolic, or structural changes in ischemic tissues resulting from the restoration of blood flow to the tissue (REPERFUSION), including swelling; HEMORRHAGE; NECROSIS; and damage from FREE RADICALS. The most common instance is MYOCARDIAL REPERFUSION INJURY.
| Descriptor ID |
D015427
|
| MeSH Number(s) |
C14.907.725 C23.550.767.877
|
| Concept/Terms |
Reperfusion Injury- Reperfusion Injury
- Reperfusion Damage
- Damage, Reperfusion
- Damages, Reperfusion
- Reperfusion Damages
- Ischemia-Reperfusion Injury
- Ischemia Reperfusion Injury
- Injury, Ischemia-Reperfusion
- Injuries, Ischemia-Reperfusion
- Injury, Ischemia Reperfusion
- Ischemia-Reperfusion Injuries
- Injury, Reperfusion
- Injuries, Reperfusion
- Reperfusion Injuries
|
Below are MeSH descriptors whose meaning is more general than "Reperfusion Injury".
Below are MeSH descriptors whose meaning is more specific than "Reperfusion Injury".
This graph shows the total number of publications written about "Reperfusion Injury" by people in this website by year, and whether "Reperfusion Injury" was a major or minor topic of these publications.
To see the data from this visualization as text,
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| Year | Major Topic | Minor Topic | Total |
|---|
| 1997 | 1 | 0 | 1 |
| 1999 | 0 | 1 | 1 |
| 2010 | 0 | 1 | 1 |
| 2012 | 0 | 1 | 1 |
| 2018 | 2 | 0 | 2 |
| 2020 | 1 | 0 | 1 |
| 2021 | 1 | 0 | 1 |
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Below are the most recent publications written about "Reperfusion Injury" by people in Profiles.
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Crosstalk between nonclassical monocytes and alveolar macrophages mediates transplant ischemia-reperfusion injury through classical monocyte recruitment. JCI Insight. 2021 03 22; 6(6).
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Residual endotoxin induces primary graft dysfunction through ischemia/reperfusion-primed alveolar macrophages. J Clin Invest. 2020 08 03; 130(8):4456-4469.
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Kidney-derived c-kit+ progenitor/stem cells contribute to podocyte recovery in a model of acute proteinuria. Sci Rep. 2018 10 03; 8(1):14723.
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Sevoflurane Protects Hepatocytes From Ischemic Injury by Reducing Reactive Oxygen Species Signaling of Hepatic Stellate Cells: Translational Findings Based on a Clinical Trial. Anesth Analg. 2018 10; 127(4):1058-1065.
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Spleen-derived classical monocytes mediate lung ischemia-reperfusion injury through IL-1?. J Clin Invest. 2018 07 02; 128(7):2833-2847.
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Inorganic polyphosphate is a potent activator of the mitochondrial permeability transition pore in cardiac myocytes. J Gen Physiol. 2012 May; 139(5):321-31.
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YSPSL (rPSGL-Ig) for improvement of early renal allograft function: a double-blind, placebo-controlled, multi-center Phase IIa study. Clin Transplant. 2011 Jul-Aug; 25(4):523-33.
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Tauroursodeoxycholate ameliorates reperfusion injury after pig liver transplantation. Transpl Int. 1999; 12(6):454-62.
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Free radical spin traps as adjuncts for the prevention and treatment of disease. Med Hypotheses. 1999 Jan; 52(1):53-7.
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Cytochrome P-450 2E1 in rat liver peroxisomes: downregulation by ischemia/reperfusion-induced oxidative stress. Free Radic Biol Med. 1997; 23(7):963-71.