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miR-132/212 deficiency impairs tau metabolism and promotes pathological aggregation in vivo.
Pathologic quiz case: a 27-week female fetus with massive macrocephaly and generalized anasarca. Congenital immature intracranial teratoma with skull rupture and high output cardiac failure.
Relationship between circulating tumor-associated autoantibodies and clinical outcomes in advanced-stage NSCLC patients receiving PD-1/-L1 directed immune checkpoint inhibition.
Spinal implant debris-induced osteolysis.
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miR-132/212 deficiency impairs tau metabolism and promotes pathological aggregation in vivo.
miR-132/212 deficiency impairs tau metabolism and promotes pathological aggregation in vivo. Hum Mol Genet. 2015 Dec 01; 24(23):6721-35.
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PubMed
subject areas
Animals
Cognition Disorders
Disease Models, Animal
Down-Regulation
Gene Expression Regulation
Glycogen Synthase Kinase 3
Glycogen Synthase Kinase 3 beta
Humans
Mice
MicroRNAs
Phosphorylation
Protein Aggregation, Pathological
tau Proteins
Tauopathies
authors with profiles
David A Bennett