NK Cell Lectin-Like Receptor Subfamily K
"NK Cell Lectin-Like Receptor Subfamily K" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
An activating NK cell lectin-like receptor subfamily that regulates immune responses to INFECTION and NEOPLASMS. Members of this subfamily generally occur as homodimers.
| Descriptor ID |
D055655
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| MeSH Number(s) |
D12.776.543.750.705.895.800.910
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| Concept/Terms |
NK Cell Lectin-Like Receptor Subfamily K- NK Cell Lectin-Like Receptor Subfamily K
- NK Cell Lectin Like Receptor Subfamily K
- Killer Cell Lectin-Like Receptor Subfamily K
- Killer Cell Lectin Like Receptor Subfamily K
NK Cell Lectin-Like Receptor Subfamily K, Member 1- NK Cell Lectin-Like Receptor Subfamily K, Member 1
- NK Cell Lectin Like Receptor Subfamily K, Member 1
- NKG2D Receptor
- Receptor, NKG2D
- CD314 Antigen
- Antigen, CD314
- NKG2-D Receptor
- NKG2 D Receptor
- Receptor, NKG2-D
- Killer Cell Lectin-Like Receptor Subfamily K, Member 1
- Killer Cell Lectin Like Receptor Subfamily K, Member 1
- NK Cell Receptor D
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Below are MeSH descriptors whose meaning is more general than "NK Cell Lectin-Like Receptor Subfamily K".
Below are MeSH descriptors whose meaning is more specific than "NK Cell Lectin-Like Receptor Subfamily K".
This graph shows the total number of publications written about "NK Cell Lectin-Like Receptor Subfamily K" by people in this website by year, and whether "NK Cell Lectin-Like Receptor Subfamily K" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2006 | 0 | 1 | 1 |
| 2007 | 0 | 2 | 2 |
| 2009 | 0 | 1 | 1 |
| 2010 | 0 | 1 | 1 |
| 2012 | 1 | 0 | 1 |
| 2016 | 1 | 0 | 1 |
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Below are the most recent publications written about "NK Cell Lectin-Like Receptor Subfamily K" by people in Profiles.
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CD155 on HIV-Infected Cells Is Not Modulated by HIV-1 Vpu and Nef but Synergizes with NKG2D Ligands to Trigger NK Cell Lysis of Autologous Primary HIV-Infected Cells. AIDS Res Hum Retroviruses. 2017 02; 33(2):93-100.
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NKG2D signaling on CD8? T cells represses T-bet and rescues CD4-unhelped CD8? T cell memory recall but not effector responses. Nat Med. 2012 Feb 26; 18(3):422-8.
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Degranulation of natural killer cells following interaction with HIV-1-infected cells is hindered by downmodulation of NTB-A by Vpu. Cell Host Microbe. 2010 Nov 18; 8(5):397-409.
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HIV-1 Vpr triggers natural killer cell-mediated lysis of infected cells through activation of the ATR-mediated DNA damage response. PLoS Pathog. 2009 Oct; 5(10):e1000613.
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Human T-cell leukemia virus type 1 (HTLV-1) p12I down-modulates ICAM-1 and -2 and reduces adherence of natural killer cells, thereby protecting HTLV-1-infected primary CD4+ T cells from autologous natural killer cell-mediated cytotoxicity despite the reduction of major histocompatibility complex class I molecules on infected cells. J Virol. 2007 Sep; 81(18):9707-17.
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HIV modulates the expression of ligands important in triggering natural killer cell cytotoxic responses on infected primary T-cell blasts. Blood. 2007 Aug 15; 110(4):1207-14.
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Clinicopathological significance of major histocompatibility complex class I-related chain a and B expression in thyroid cancer. J Clin Endocrinol Metab. 2006 Jul; 91(7):2704-12.