"Monocytes" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Large, phagocytic mononuclear leukocytes produced in the vertebrate BONE MARROW and released into the BLOOD; contain a large, oval or somewhat indented nucleus surrounded by voluminous cytoplasm and numerous organelles.
| Descriptor ID |
D009000
|
| MeSH Number(s) |
A11.118.637.555.652 A11.148.580 A11.627.624 A11.733.547 A15.145.229.637.555.652 A15.378.316.580 A15.382.490.555.652 A15.382.680.547 A15.382.812.547
|
| Concept/Terms |
|
Below are MeSH descriptors whose meaning is more general than "Monocytes".
Below are MeSH descriptors whose meaning is more specific than "Monocytes".
This graph shows the total number of publications written about "Monocytes" by people in this website by year, and whether "Monocytes" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1997 | 1 | 1 | 2 |
| 1998 | 1 | 0 | 1 |
| 2017 | 1 | 0 | 1 |
To return to the timeline,
click here.
Below are the most recent publications written about "Monocytes" by people in Profiles.
-
Tissue-resident, extravascular Ly6c- monocytes are critical for inflammation in the synovium. Cell Rep. 2023 05 30; 42(5):112513.
-
Crosstalk between nonclassical monocytes and alveolar macrophages mediates transplant ischemia-reperfusion injury through classical monocyte recruitment. JCI Insight. 2021 03 22; 6(6).
-
Spleen-derived classical monocytes mediate lung ischemia-reperfusion injury through IL-1?. J Clin Invest. 2018 07 02; 128(7):2833-2847.
-
Wnt7a induces a unique phenotype of monocyte-derived macrophages with lower phagocytic capacity and differential expression of pro- and anti-inflammatory cytokines. Immunology. 2018 02; 153(2):203-213.
-
Donor pulmonary intravascular nonclassical monocytes recruit recipient neutrophils and mediate primary lung allograft dysfunction. Sci Transl Med. 2017 06 14; 9(394).
-
CD33 Alzheimer's disease locus: altered monocyte function and amyloid biology. Nat Neurosci. 2013 Jul; 16(7):848-50.
-
The cAMP-dependent protein kinase A and protein kinase C-beta pathways synergistically interact to activate HIV-1 transcription in latently infected cells of monocyte/macrophage lineage. Virology. 1998 Jun 05; 245(2):257-69.
-
TNFalpha cooperates with the protein kinase A pathway to synergistically increase HIV-1 LTR transcription via downstream TRE-like cAMP response elements. Virology. 1997 Oct 27; 237(2):422-9.
-
A potent activator of HIV-1 replication is present in the genital tract of a subset of HIV-1-infected and uninfected women. AIDS. 1997 Sep; 11(11):1319-26.