Endoplasmic Reticulum Stress
"Endoplasmic Reticulum Stress" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Various physiological or molecular disturbances that impair ENDOPLASMIC RETICULUM function. It triggers many responses, including UNFOLDED PROTEIN RESPONSE, which may lead to APOPTOSIS; and AUTOPHAGY.
Descriptor ID |
D059865
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MeSH Number(s) |
G04.434
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Concept/Terms |
Endoplasmic Reticulum Stress- Endoplasmic Reticulum Stress
- Endoplasmic Reticulum Stresses
- Reticulum Stress, Endoplasmic
- Reticulum Stresses, Endoplasmic
- Stresses, Endoplasmic Reticulum
- Stress, Endoplasmic Reticulum
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Below are MeSH descriptors whose meaning is more general than "Endoplasmic Reticulum Stress".
Below are MeSH descriptors whose meaning is more specific than "Endoplasmic Reticulum Stress".
This graph shows the total number of publications written about "Endoplasmic Reticulum Stress" by people in this website by year, and whether "Endoplasmic Reticulum Stress" was a major or minor topic of these publications.
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Year | Major Topic | Minor Topic | Total |
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2015 | 0 | 1 | 1 |
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Below are the most recent publications written about "Endoplasmic Reticulum Stress" by people in Profiles.
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ER stress sensor, glucose regulatory protein 78 (GRP78) regulates redox status in pancreatic cancer thereby maintaining "stemness". Cell Death Dis. 2019 02 12; 10(2):132.
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Type 2 deiodinase polymorphism causes ER stress and hypothyroidism in the brain. J Clin Invest. 2019 01 02; 129(1):230-245.
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The Deiodinase Trio and Thyroid Hormone Signaling. Methods Mol Biol. 2018; 1801:67-83.
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Podocytes exhibit a specialized protein quality control employing derlin-2 in kidney disease. Am J Physiol Renal Physiol. 2018 03 01; 314(3):F471-F482.
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High fat-diet and saturated fatty acid palmitate inhibits IGF-1 function in chondrocytes. Osteoarthritis Cartilage. 2017 09; 25(9):1516-1521.
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Inhibition of Sp1 prevents ER homeostasis and causes cell death by lysosomal membrane permeabilization in pancreatic cancer. Sci Rep. 2017 05 08; 7(1):1564.
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Defective podocyte insulin signalling through p85-XBP1 promotes ATF6-dependent maladaptive ER-stress response in diabetic nephropathy. Nat Commun. 2015 Mar 10; 6:6496.
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ATF4 Gene Network Mediates Cellular Response to the Anticancer PAD Inhibitor YW3-56 in Triple-Negative Breast Cancer Cells. Mol Cancer Ther. 2015 Apr; 14(4):877-88.
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Endoplasmic reticulum stress decreases intracellular thyroid hormone activation via an eIF2a-mediated decrease in type 2 deiodinase synthesis. Mol Endocrinol. 2011 Dec; 25(12):2065-75.