 NK Cell Lectin-Like Receptor Subfamily K
"NK Cell Lectin-Like Receptor Subfamily K" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
An activating NK cell lectin-like receptor subfamily that regulates immune responses to INFECTION and NEOPLASMS. Members of this subfamily generally occur as homodimers.
| Descriptor ID |
D055655
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| MeSH Number(s) |
D12.776.543.750.705.895.800.910 D23.050.301.264.035.582 D23.101.100.110.582
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| Concept/Terms |
NK Cell Lectin-Like Receptor Subfamily K- NK Cell Lectin-Like Receptor Subfamily K
- NK Cell Lectin Like Receptor Subfamily K
- Killer Cell Lectin-Like Receptor Subfamily K
- Killer Cell Lectin Like Receptor Subfamily K
NK Cell Lectin-Like Receptor Subfamily K, Member 1- NK Cell Lectin-Like Receptor Subfamily K, Member 1
- NK Cell Lectin Like Receptor Subfamily K, Member 1
- NKG2D Receptor
- Receptor, NKG2D
- NK Cell Receptor D
- NKG2-D Receptor
- NKG2 D Receptor
- Receptor, NKG2-D
- CD314 Antigen
- Antigen, CD314
- Killer Cell Lectin-Like Receptor Subfamily K, Member 1
- Killer Cell Lectin Like Receptor Subfamily K, Member 1
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Below are MeSH descriptors whose meaning is more general than "NK Cell Lectin-Like Receptor Subfamily K".
- Chemicals and Drugs [D]
- Amino Acids, Peptides, and Proteins [D12]
- Proteins [D12.776]
- Membrane Proteins [D12.776.543]
- Receptors, Cell Surface [D12.776.543.750]
- Receptors, Immunologic [D12.776.543.750.705]
- Receptors, Natural Killer Cell [D12.776.543.750.705.895]
- Receptors, NK Cell Lectin-Like [D12.776.543.750.705.895.800]
- NK Cell Lectin-Like Receptor Subfamily K [D12.776.543.750.705.895.800.910]
- Biological Factors [D23]
- Antigens [D23.050]
- Antigens, Surface [D23.050.301]
- Antigens, Differentiation [D23.050.301.264]
- Antigens, CD [D23.050.301.264.035]
- NK Cell Lectin-Like Receptor Subfamily K [D23.050.301.264.035.582]
- Biological Markers [D23.101]
- Antigens, Differentiation [D23.101.100]
- Antigens, CD [D23.101.100.110]
- NK Cell Lectin-Like Receptor Subfamily K [D23.101.100.110.582]
Below are MeSH descriptors whose meaning is more specific than "NK Cell Lectin-Like Receptor Subfamily K".
This graph shows the total number of publications written about "NK Cell Lectin-Like Receptor Subfamily K" by people in this website by year, and whether "NK Cell Lectin-Like Receptor Subfamily K" was a major or minor topic of these publications.
To see the data from this visualization as text, click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2006 | 0 | 1 | 1 | | 2007 | 0 | 2 | 2 | | 2009 | 0 | 1 | 1 | | 2010 | 0 | 1 | 1 | | 2012 | 1 | 0 | 1 | | 2016 | 1 | 0 | 1 |
To return to the timeline, click here.
Below are the most recent publications written about "NK Cell Lectin-Like Receptor Subfamily K" by people in Profiles.
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Davis ZB, Sowrirajan B, Cogswell A, Ward JP, Planelles V, Barker E. CD155 on HIV-Infected Cells Is Not Modulated by HIV-1 Vpu and Nef but Synergizes with NKG2D Ligands to Trigger NK Cell Lysis of Autologous Primary HIV-Infected Cells. AIDS Res Hum Retroviruses. 2017 02; 33(2):93-100.
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Zloza A, Kohlhapp FJ, Lyons GE, Schenkel JM, Moore TV, Lacek AT, O'Sullivan JA, Varanasi V, Williams JW, Jagoda MC, Bellavance EC, Marzo AL, Thomas PG, Zafirova B, Polic B, Al-Harthi L, Sperling AI, Guevara-Patiño JA. NKG2D signaling on CD8? T cells represses T-bet and rescues CD4-unhelped CD8? T cell memory recall but not effector responses. Nat Med. 2012 Feb 26; 18(3):422-8.
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Shah AH, Sowrirajan B, Davis ZB, Ward JP, Campbell EM, Planelles V, Barker E. Degranulation of natural killer cells following interaction with HIV-1-infected cells is hindered by downmodulation of NTB-A by Vpu. Cell Host Microbe. 2010 Nov 18; 8(5):397-409.
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Ward J, Davis Z, DeHart J, Zimmerman E, Bosque A, Brunetta E, Mavilio D, Planelles V, Barker E. HIV-1 Vpr triggers natural killer cell-mediated lysis of infected cells through activation of the ATR-mediated DNA damage response. PLoS Pathog. 2009 Oct; 5(10):e1000613.
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Banerjee P, Feuer G, Barker E. Human T-cell leukemia virus type 1 (HTLV-1) p12I down-modulates ICAM-1 and -2 and reduces adherence of natural killer cells, thereby protecting HTLV-1-infected primary CD4+ T cells from autologous natural killer cell-mediated cytotoxicity despite the reduction of major histocompatibility complex class I molecules on infected cells. J Virol. 2007 Sep; 81(18):9707-17.
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Ward J, Bonaparte M, Sacks J, Guterman J, Fogli M, Mavilio D, Barker E. HIV modulates the expression of ligands important in triggering natural killer cell cytotoxic responses on infected primary T-cell blasts. Blood. 2007 Aug 15; 110(4):1207-14.
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Xu X, Rao G, Gaffud MJ, Ding HG, Maki G, Klingemann HG, Groh V, Spies T, Caillat-Zucman S, Gattuso P, Plate J, Prinz RA. Clinicopathological significance of major histocompatibility complex class I-related chain a and B expression in thyroid cancer. J Clin Endocrinol Metab. 2006 Jul; 91(7):2704-12.
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