 Prenatal Exposure Delayed Effects
"Prenatal Exposure Delayed Effects" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
The consequences of exposing the FETUS in utero to certain factors, such as NUTRITION PHYSIOLOGICAL PHENOMENA; PHYSIOLOGICAL STRESS; DRUGS; RADIATION; and other physical or chemical factors. These consequences are observed later in the offspring after BIRTH.
| Descriptor ID |
D011297
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| MeSH Number(s) |
C13.703.824.500
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| Concept/Terms |
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Below are MeSH descriptors whose meaning is more general than "Prenatal Exposure Delayed Effects".
Below are MeSH descriptors whose meaning is more specific than "Prenatal Exposure Delayed Effects".
This graph shows the total number of publications written about "Prenatal Exposure Delayed Effects" by people in this website by year, and whether "Prenatal Exposure Delayed Effects" was a major or minor topic of these publications.
To see the data from this visualization as text, click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1994 | 2 | 0 | 2 | | 1996 | 2 | 0 | 2 | | 1998 | 2 | 0 | 2 | | 2002 | 2 | 0 | 2 | | 2003 | 3 | 0 | 3 | | 2004 | 2 | 0 | 2 | | 2006 | 1 | 0 | 1 | | 2007 | 1 | 0 | 1 | | 2008 | 1 | 0 | 1 | | 2009 | 1 | 0 | 1 | | 2010 | 1 | 0 | 1 | | 2011 | 1 | 0 | 1 | | 2013 | 1 | 0 | 1 | | 2019 | 2 | 0 | 2 | | 2021 | 0 | 1 | 1 |
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Below are the most recent publications written about "Prenatal Exposure Delayed Effects" by people in Profiles.
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Assadi F, Mazaheri M. Long-term cardiometabolic consequences among adolescent offspring born to women with type1 diabetes. Prim Care Diabetes. 2022 02; 16(1):122-126.
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Gluncic V, Moric M, Chu Y, Hanko V, Li J, Lukic IK, Lukic A, Edassery SL, Kroin JS, Persons AL, Perry P, Kelly L, Shiveley TJ, Nice K, Napier TC, Kordower JH, Tuman KJ. In utero Exposure to Anesthetics Alters Neuronal Migration Pattern in Developing Cerebral Cortex and Causes Postnatal Behavioral Deficits in Rats. Cereb Cortex. 2019 12 17; 29(12):5285-5301.
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Hu Z, Tylavsky FA, Han JC, Kocak M, Fowke JH, Davis RL, Lewinn K, Bush NR, Zhao Q. Maternal metabolic factors during pregnancy predict early childhood growth trajectories and obesity risk: the CANDLE Study. Int J Obes (Lond). 2019 10; 43(10):1914-1922.
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Parsons EC, Patel K, Tran BT, Littman AJ. Maternal pre-gravid obesity and early childhood respiratory hospitalization: a population-based case-control study. Matern Child Health J. 2013 Aug; 17(6):1095-102.
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Thompson VB, Koprich JB, Chen EY, Kordower JH, Terpstra BT, Lipton JW. Prenatal exposure to MDMA alters noradrenergic neurodevelopment in the rat. Neurotoxicol Teratol. 2012 Jan-Feb; 34(1):206-13.
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Miodovnik A, Engel SM, Zhu C, Ye X, Soorya LV, Silva MJ, Calafat AM, Wolff MS. Endocrine disruptors and childhood social impairment. Neurotoxicology. 2011 Mar; 32(2):261-7.
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Wang S, Yan JY, Lo YK, Carvey PM, Ling Z. Dopaminergic and serotoninergic deficiencies in young adult rats prenatally exposed to the bacterial lipopolysaccharide. Brain Res. 2009 Apr 10; 1265:196-204.
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Ling Z, Zhu Y, Tong CW, Snyder JA, Lipton JW, Carvey PM. Prenatal lipopolysaccharide does not accelerate progressive dopamine neuron loss in the rat as a result of normal aging. Exp Neurol. 2009 Apr; 216(2):312-20.
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Zhu Y, Carvey PM, Ling Z. Altered glutathione homeostasis in animals prenatally exposed to lipopolysaccharide. Neurochem Int. 2007 Mar; 50(4):671-80.
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Ling Z, Zhu Y, Tong Cw, Snyder JA, Lipton JW, Carvey PM. Progressive dopamine neuron loss following supra-nigral lipopolysaccharide (LPS) infusion into rats exposed to LPS prenatally. Exp Neurol. 2006 Jun; 199(2):499-512.
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