Major Histocompatibility Complex
"Major Histocompatibility Complex" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
The genetic region which contains the loci of genes which determine the structure of the serologically defined (SD) and lymphocyte-defined (LD) TRANSPLANTATION ANTIGENS, genes which control the structure of the IMMUNE RESPONSE-ASSOCIATED ANTIGENS, HUMAN; the IMMUNE RESPONSE GENES which control the ability of an animal to respond immunologically to antigenic stimuli, and genes which determine the structure and/or level of the first four components of complement.
| Descriptor ID |
D008285
|
| MeSH Number(s) |
G05.360.340.024.340.610 G05.360.340.024.380.500 G12.500.500
|
| Concept/Terms |
Major Histocompatibility Complex- Major Histocompatibility Complex
- Complex, Major Histocompatibility
- Complices, Major Histocompatibility
- Histocompatibility Complex, Major
- Histocompatibility Complices, Major
- Major Histocompatibility Complices
- Histocompatibility Complex
- Complex, Histocompatibility
- Complices, Histocompatibility
- Histocompatibility Complices
|
Below are MeSH descriptors whose meaning is more general than "Major Histocompatibility Complex".
Below are MeSH descriptors whose meaning is more specific than "Major Histocompatibility Complex".
This graph shows the total number of publications written about "Major Histocompatibility Complex" by people in this website by year, and whether "Major Histocompatibility Complex" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2004 | 0 | 1 | 1 |
| 2006 | 0 | 1 | 1 |
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Below are the most recent publications written about "Major Histocompatibility Complex" by people in Profiles.
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An Arthritis-Suppressive and Treg Cell-Inducing CD4+ T Cell Epitope Is Functional in the Context of HLA-Restricted T Cell Responses. Arthritis Rheumatol. 2016 Mar; 68(3):639-47.
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Non-MHC risk alleles in rheumatoid arthritis and in the syntenic chromosome regions of corresponding animal models. Clin Dev Immunol. 2012; 2012:284751.
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Experimental spondyloarthropathies: animal models of ankylosing spondylitis. Curr Rheumatol Rep. 2006 Aug; 8(4):267-74.
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Camptocormia associated with focal myositis in multiple-system atrophy. Mov Disord. 2006 Mar; 21(3):390-4.
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Killing of human immunodeficiency virus-infected primary T-cell blasts by autologous natural killer cells is dependent on the ability of the virus to alter the expression of major histocompatibility complex class I molecules. Blood. 2004 Oct 01; 104(7):2087-94.
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Disease-associated qualitative and quantitative trait loci in proteoglycan-induced arthritis and collagen-induced arthritis. Am J Med Sci. 2004 Apr; 327(4):188-95.
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Study of antigen-processing steps reveals preferences explaining differential biological outcomes of two HLA-A2-restricted immunodominant epitopes from human immunodeficiency virus type 1. J Virol. 2002 Oct; 76(20):10219-25.
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Major histocompatibility complex controls susceptibility and dominant inheritance, but not the severity of the disease in mouse models of rheumatoid arthritis. Immunogenetics. 2002 Jun; 54(3):184-92.
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Human antibodies to major histocompatibility complex alloantigens mediate lysis and neutralization of HIV-1 primary isolate virions in the presence of complement. J Acquir Immune Defic Syndr. 2001 Feb 01; 26(2):103-10.
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A proteoglycan (aggrecan)-specific T cell hybridoma induces arthritis in BALB/c mice. J Immunol. 1995 Sep 01; 155(5):2679-87.