Membrane Potential, Mitochondrial
"Membrane Potential, Mitochondrial" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
The voltage difference, normally maintained at approximately -180mV, across the INNER MITOCHONDRIAL MEMBRANE, by a net movement of positive charge across the membrane. It is a major component of the PROTON MOTIVE FORCE in MITOCHONDRIA used to drive the synthesis of ATP.
| Descriptor ID |
D053078
|
| MeSH Number(s) |
G03.295.770.500 G04.580.550 G07.265.675.550
|
| Concept/Terms |
Membrane Potential, Mitochondrial- Membrane Potential, Mitochondrial
- Membrane Potentials, Mitochondrial
- Mitochondrial Membrane Potentials
- Potential, Mitochondrial Membrane
- Potentials, Mitochondrial Membrane
- Mitochondrial Membrane Potential
|
Below are MeSH descriptors whose meaning is more general than "Membrane Potential, Mitochondrial".
Below are MeSH descriptors whose meaning is more specific than "Membrane Potential, Mitochondrial".
This graph shows the total number of publications written about "Membrane Potential, Mitochondrial" by people in this website by year, and whether "Membrane Potential, Mitochondrial" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2007 | 0 | 1 | 1 |
| 2009 | 0 | 2 | 2 |
| 2011 | 0 | 1 | 1 |
| 2012 | 0 | 1 | 1 |
| 2013 | 0 | 2 | 2 |
| 2014 | 0 | 1 | 1 |
| 2015 | 0 | 1 | 1 |
| 2017 | 0 | 2 | 2 |
| 2020 | 0 | 1 | 1 |
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Below are the most recent publications written about "Membrane Potential, Mitochondrial" by people in Profiles.
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Amorphous SiO2 nanoparticles promote cardiac dysfunction via the opening of the mitochondrial permeability transition pore in rat heart and human cardiomyocytes. Part Fibre Toxicol. 2020 May 07; 17(1):15.
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Small Interfering RNA Targeting Mitochondrial Calcium Uniporter Improves Cardiomyocyte Cell Viability in Hypoxia/Reoxygenation Injury by Reducing Calcium Overload. Oxid Med Cell Longev. 2017; 2017:5750897.
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Silica nanoparticles induce cardiotoxicity interfering with energetic status and Ca2+ handling in adult rat cardiomyocytes. Am J Physiol Heart Circ Physiol. 2017 Apr 01; 312(4):H645-H661.
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Inositol 1,4,5-trisphosphate-mediated sarcoplasmic reticulum-mitochondrial crosstalk influences adenosine triphosphate production via mitochondrial Ca2+ uptake through the mitochondrial ryanodine receptor in cardiac myocytes. Cardiovasc Res. 2016 10; 112(1):491-501.
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Distinct mPTP activation mechanisms in ischaemia-reperfusion: contributions of Ca2+, ROS, pH, and inorganic polyphosphate. Cardiovasc Res. 2015 May 01; 106(2):237-48.
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Short communication: Apoptosis pathways in HIV-1-infected patients before and after highly active antiretroviral therapy: relevance to immune recovery. AIDS Res Hum Retroviruses. 2015 Feb; 31(2):208-16.
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Ischemia/Reperfusion injury protection by mesenchymal stem cell derived antioxidant capacity. Stem Cells Dev. 2013 Sep 15; 22(18):2497-507.
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Effects of mitochondrial uncoupling on Ca(2+) signaling during excitation-contraction coupling in atrial myocytes. Am J Physiol Heart Circ Physiol. 2013 Apr 01; 304(7):H983-93.
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Inorganic polyphosphate is a potent activator of the mitochondrial permeability transition pore in cardiac myocytes. J Gen Physiol. 2012 May; 139(5):321-31.
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Measuring mitochondrial function in intact cardiac myocytes. J Mol Cell Cardiol. 2012 Jan; 52(1):48-61.