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One or more keywords matched the following properties of Kanaporis, Giedrius
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overview My Scopus ID is 21742760800. My NIH COMMONS name is GKANAPORIS. Research Areas: Cardiac electrophysiology, cardiac arrhythmias, heart failure, intracellular calcium signaling in subcellular domains, cell-to-cell coupling. My Faculty Profile at Rush University Medical Center: https://www.rushu.rush.edu/faculty/giedrius-kanaporis-phd My Laboratory: https://www.rushu.rush.edu/research/departmental-research/physiology-and-biophysics-research/laboratory-lothar-blatter-md-dr-med My NCBI Bibliography: https://www.ncbi.nlm.nih.gov/myncbi/browse/collection/48071495/?sort=date&direction=descending My Scopus: https://www.scopus.com/authid/detail.uri?authorId=21742760800 Education: PhD, Kaunas University of Medicine, Kaunas, Lithuania MS, Vytautas Magnus University, Kaunas, Lithuania BS, Vytautas Magnus University, Kaunas, Lithuania
One or more keywords matched the following items that are connected to Kanaporis, Giedrius
Item TypeName
Concept Heart Atria
Concept Heart Conduction System
Concept Heart
Concept Heart Ventricles
Concept Heart Failure
Concept Heart Rate
Academic Article Effect of hyperosmolarity on beta2-adrenergic stimulation in human atrium.
Academic Article Changes of beta2-adrenergic stimulation induced by hyperosmosis in human atrium.
Academic Article Optical mapping at increased illumination intensities.
Academic Article The mechanisms of calcium cycling and action potential dynamics in cardiac alternans.
Academic Article Calcium-activated chloride current determines action potential morphology during calcium alternans in atrial myocytes.
Academic Article Ca(2+)-activated chloride channel activity during Ca(2+) alternans in ventricular myocytes.
Academic Article Membrane potential determines calcium alternans through modulation of SR Ca2+ load and L-type Ca2+ current.
Academic Article Alternans in atria: Mechanisms and clinical relevance.
Academic Article AP and Ca2+ alternans: An inseparable couple.
Academic Article Metabolic inhibition reduces cardiac L-type Ca2+ channel current due to acidification caused by ATP hydrolysis.
Academic Article Action potential shortening rescues atrial calcium alternans.
Academic Article Triggered Ca2+ Waves Induce Depolarization of Maximum Diastolic Potential and Action Potential Prolongation in Dog Atrial Myocytes.
Academic Article Calcium- and voltage-driven atrial alternans: Insight from [Ca]i and Vm asynchrony.
Academic Article Increased Risk for Atrial Alternans in Rabbit Heart Failure: The Role of Ca2+/Calmodulin-Dependent Kinase II and Inositol-1,4,5-trisphosphate Signaling.
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