Sebastian Pollandt to Long-Term Potentiation
This is a "connection" page, showing publications Sebastian Pollandt has written about Long-Term Potentiation.
Connection Strength
0.914
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Cocaine withdrawal enhances long-term potentiation induced by corticotropin-releasing factor at central amygdala glutamatergic synapses via CRF, NMDA receptors and PKA. Eur J Neurosci. 2006 Sep; 24(6):1733-43.
Score: 0.278
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8-OH-DPAT suppresses the induction of LTP in brain slices of the rat lateral amygdala. Neuroreport. 2003 May 06; 14(6):895-7.
Score: 0.221
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Fear potentiated startle increases phospholipase D (PLD) expression/activity and PLD-linked metabotropic glutamate receptor mediated post-tetanic potentiation in rat amygdala. Neurobiol Learn Mem. 2016 Feb; 128:65-79.
Score: 0.133
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Cocaine withdrawal reduces group I mGluR-mediated long-term potentiation via decreased GABAergic transmission in the amygdala. Eur J Neurosci. 2011 Jul; 34(2):177-89.
Score: 0.097
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Dopamine receptor mechanisms mediate corticotropin-releasing factor-induced long-term potentiation in the rat amygdala following cocaine withdrawal. Eur J Neurosci. 2010 Mar; 31(6):1027-42.
Score: 0.089
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Long-term potentiation (LTP) in the central amygdala (CeA) is enhanced after prolonged withdrawal from chronic cocaine and requires CRF1 receptors. J Neurophysiol. 2007 Jan; 97(1):937-41.
Score: 0.070
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Dopamine-induced plasticity, phospholipase D (PLD) activity and cocaine-cue behavior depend on PLD-linked metabotropic glutamate receptors in amygdala. PLoS One. 2011; 6(9):e25639.
Score: 0.025