 Sympatholytics
"Sympatholytics" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Drugs that inhibit the actions of the sympathetic nervous system by any mechanism. The most common of these are the ADRENERGIC ANTAGONISTS and drugs that deplete norepinephrine or reduce the release of transmitters from adrenergic postganglionic terminals (see ADRENERGIC AGENTS). Drugs that act in the central nervous system to reduce sympathetic activity (e.g., centrally acting alpha-2 adrenergic agonists, see ADRENERGIC ALPHA-AGONISTS) are included here.
| Descriptor ID |
D013565
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| MeSH Number(s) |
D27.505.696.663.050.850
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| Concept/Terms |
Sympatholytics- Sympatholytics
- Sympatholytic Agents
- Agents, Sympatholytic
- Sympatholytic Drugs
- Drugs, Sympatholytic
- Sympathetic-Blocking Agents
- Agents, Sympathetic-Blocking
- Sympathetic Blocking Agents
Sympatholytic Effect- Sympatholytic Effect
- Effect, Sympatholytic
- Sympatholytic Effects
- Effects, Sympatholytic
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Below are MeSH descriptors whose meaning is more general than "Sympatholytics".
Below are MeSH descriptors whose meaning is more specific than "Sympatholytics".
This graph shows the total number of publications written about "Sympatholytics" by people in this website by year, and whether "Sympatholytics" was a major or minor topic of these publications.
To see the data from this visualization as text, click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1996 | 1 | 1 | 2 | | 1999 | 0 | 1 | 1 | | 2004 | 0 | 1 | 1 | | 2005 | 0 | 1 | 1 | | 2007 | 0 | 1 | 1 | | 2008 | 0 | 2 | 2 | | 2011 | 0 | 1 | 1 |
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Below are the most recent publications written about "Sympatholytics" by people in Profiles.
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Kordower JH, Dodiya HB, Kordower AM, Terpstra B, Paumier K, Madhavan L, Sortwell C, Steece-Collier K, Collier TJ. Transfer of host-derived a synuclein to grafted dopaminergic neurons in rat. Neurobiol Dis. 2011 Sep; 43(3):552-7.
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Isacson O, Kordower JH. Future of cell and gene therapies for Parkinson's disease. Ann Neurol. 2008 Dec; 64 Suppl 2:S122-38.
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Turner MS, Gray TS, Mickiewicz AL, Napier TC. Fos expression following activation of the ventral pallidum in normal rats and in a model of Parkinson's Disease: implications for limbic system and basal ganglia interactions. Brain Struct Funct. 2008 Sep; 213(1-2):197-213.
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Gasmi M, Brandon EP, Herzog CD, Wilson A, Bishop KM, Hofer EK, Cunningham JJ, Printz MA, Kordower JH, Bartus RT. AAV2-mediated delivery of human neurturin to the rat nigrostriatal system: long-term efficacy and tolerability of CERE-120 for Parkinson's disease. Neurobiol Dis. 2007 Jul; 27(1):67-76.
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Maries E, Kordower JH, Chu Y, Collier TJ, Sortwell CE, Olaru E, Shannon K, Steece-Collier K. Focal not widespread grafts induce novel dyskinetic behavior in parkinsonian rats. Neurobiol Dis. 2006 Jan; 21(1):165-80.
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Heidenreich BA, Mitrovic I, Battaglia G, Napier TC. Limbic pallidal adaptations following long-term cessation of dopaminergic transmission: lack of upregulation of dopamine receptor function. Exp Neurol. 2004 Apr; 186(2):145-57.
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Dodel RC, Du Y, Bales KR, Ling Z, Carvey PM, Paul SM. Caspase-3-like proteases and 6-hydroxydopamine induced neuronal cell death. Brain Res Mol Brain Res. 1999 Jan 22; 64(1):141-8.
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Carvey PM, Lin DH, Faselis CJ, Notermann JK, Ling ZD. Loss of striatal DA innervation increases striatal trophic activity directed at DA neurons in culture. Exp Neurol. 1996 Aug; 140(2):184-97.
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Kroin JS, McCarthy RJ, Penn RD, Lubenow TR, Ivankovich AD. Intrathecal clonidine and tizanidine in conscious dogs: comparison of analgesic and hemodynamic effects. Anesth Analg. 1996 Mar; 82(3):627-35.
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