"Lung Injury" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Damage to any compartment of the lung caused by physical, chemical, or biological agents which characteristically elicit inflammatory reaction. These inflammatory reactions can either be acute and dominated by NEUTROPHILS, or chronic and dominated by LYMPHOCYTES and MACROPHAGES.
| Descriptor ID |
D055370
|
| MeSH Number(s) |
C08.381.520 C26.891.554
|
| Concept/Terms |
Lung Injury- Lung Injury
- Injuries, Lung
- Injury, Lung
- Pulmonary Injury
- Injuries, Pulmonary
- Injury, Pulmonary
- Pulmonary Injuries
- Lung Injuries
Chronic Lung Injury- Chronic Lung Injury
- Chronic Lung Injuries
- Lung Injuries, Chronic
- Lung Injury, Chronic
|
Below are MeSH descriptors whose meaning is more general than "Lung Injury".
Below are MeSH descriptors whose meaning is more specific than "Lung Injury".
This graph shows the total number of publications written about "Lung Injury" by people in this website by year, and whether "Lung Injury" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 1998 | 0 | 1 | 1 |
| 2002 | 0 | 1 | 1 |
| 2013 | 0 | 2 | 2 |
| 2017 | 1 | 0 | 1 |
| 2018 | 1 | 0 | 1 |
| 2020 | 2 | 0 | 2 |
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Below are the most recent publications written about "Lung Injury" by people in Profiles.
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Residual endotoxin induces primary graft dysfunction through ischemia/reperfusion-primed alveolar macrophages. J Clin Invest. 2020 08 03; 130(8):4456-4469.
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Vaping and Use of E-Cigarette Products in Adolescents: A New Cardiopulmonary Crisis. J Cardiovasc Nurs. 2020 May/Jun; 35(3):225-228.
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Spleen-derived classical monocytes mediate lung ischemia-reperfusion injury through IL-1?. J Clin Invest. 2018 07 02; 128(7):2833-2847.
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Lung Injury and Loss of Regulatory T Cells Primes for Lung-Restricted Autoimmunity. Crit Rev Immunol. 2017; 37(1):23-37.
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Conditional deletion of FAK in mice endothelium disrupts lung vascular barrier function due to destabilization of RhoA and Rac1 activities. Am J Physiol Lung Cell Mol Physiol. 2013 Aug 15; 305(4):L291-300.
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In vivo knockdown of intersectin-1s alters endothelial cell phenotype and causes microvascular remodeling in the mouse lungs. Apoptosis. 2013 Jan; 18(1):57-76.
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Potential genetic therapies for acute lung injury. Curr Gene Ther. 2004 Dec; 4(4):487-95.
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Nitric oxide stimulates macrophage inflammatory protein-2 expression in sepsis. J Immunol. 2002 Aug 15; 169(4):2093-101.
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The relation of pneumothorax and other air leaks to mortality in the acute respiratory distress syndrome. N Engl J Med. 1998 Feb 05; 338(6):341-6.
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The adult respiratory distress syndrome. Med Clin North Am. 1983 May; 67(3):685-700.