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Jacobs, Joshua
One or more keywords matched the following items that are connected to Jacobs, Joshua
Item TypeName
Concept Cytokines
Academic Article Lymphocyte responses in patients with total hip arthroplasty.
Academic Article Concentration- and composition-dependent effects of metal ions on human MG-63 osteoblasts.
Academic Article The pathology of orthopedic implant failure is mediated by innate immune system cytokines.
Academic Article Soluble ions more than particulate cobalt-alloy implant debris induce monocyte costimulatory molecule expression and release of proinflammatory cytokines critical to metal-induced lymphocyte reactivity.
Academic Article Cytokine-controlled RANKL and osteoprotegerin expression by human and mouse synovial fibroblasts: fibroblast-mediated pathologic bone resorption.
Academic Article The role of fibroblasts and fibroblast-derived factors in periprosthetic osteolysis.
Academic Article Human monocyte response to particulate biomaterials generated in vivo and in vitro.
Academic Article Cellular mediators secreted by interfacial membranes obtained at revision total hip arthroplasty.
Academic Article New horizons in orthopaedic research: elucidation of cellular signal transduction pathways.
Academic Article Particulate wear debris activates protein tyrosine kinases and nuclear factor kappaB, which down-regulates type I collagen synthesis in human osteoblasts.
Academic Article A triple assay technique for the evaluation of metal-induced, delayed-type hypersensitivity responses in patients with or receiving total joint arthroplasty.
Academic Article The potential role of the osteoblast in the development of periprosthetic osteolysis: review of in vitro osteoblast responses to wear debris, corrosion products, and cytokines and growth factors.
Academic Article Osteolysis: basic science.
Academic Article Disease-associated qualitative and quantitative trait loci in proteoglycan-induced arthritis and collagen-induced arthritis.
Academic Article Transition from metal-DTH resistance to susceptibility is facilitated by NLRP3 inflammasome signaling induced Th17 reactivity: Implications for orthopedic implants.
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  • Cytokines