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Wei, Changli (David)
One or more keywords matched the following items that are connected to Wei, Changli (David)
Item TypeName
Concept Podocytes
Academic Article Induction of TRPC6 channel in acquired forms of proteinuric kidney disease.
Academic Article Recurrent Primary Focal Segmental Glomerulosclerosis Managed With Intensified Plasma Exchange and Concomitant Monitoring of Soluble Urokinase-Type Plasminogen Activator Receptor-Mediated Podocyte ?3-integrin Activation.
Academic Article Podocyte effacement closely links to suPAR levels at time of posttransplantation focal segmental glomerulosclerosis occurrence and improves with therapy.
Academic Article Reduction of proteinuria through podocyte alkalinization.
Academic Article Proteolytic processing of dynamin by cytoplasmic cathepsin L is a mechanism for proteinuric kidney disease.
Academic Article A Podocyte-Based Automated Screening Assay Identifies Protective Small Molecules.
Academic Article Modification of kidney barrier function by the urokinase receptor.
Academic Article CD2AP in mouse and human podocytes controls a proteolytic program that regulates cytoskeletal structure and cellular survival.
Academic Article Podocytes exhibit a specialized protein quality control employing derlin-2 in kidney disease.
Academic Article uPAR isoform 2 forms a dimer and induces severe kidney disease in mice.
Academic Article Soluble Urokinase Receptor and Acute Kidney Injury.
Academic Article The small GTPase regulatory protein Rac1 drives podocyte injury independent of cationic channel protein TRPC5.
Academic Article SuPAR mediates viral response proteinuria by rapidly changing podocyte function.
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