Changli (David) Wei to Mice, Transgenic
This is a "connection" page, showing publications Changli (David) Wei has written about Mice, Transgenic.
Connection Strength
0.392
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uPAR isoform 2 forms a dimer and induces severe kidney disease in mice. J Clin Invest. 2019 04 02; 129(5):1946-1959.
Score: 0.155
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suPAR links a dysregulated immune response to tissue inflammation and sepsis-induced acute kidney injury. JCI Insight. 2023 04 10; 8(7).
Score: 0.051
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The small GTPase regulatory protein Rac1 drives podocyte injury independent of cationic channel protein TRPC5. Kidney Int. 2023 06; 103(6):1056-1062.
Score: 0.051
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Soluble Urokinase Receptor and Acute Kidney Injury. N Engl J Med. 2020 01 30; 382(5):416-426.
Score: 0.041
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Runx2 is required for postnatal intervertebral disc tissue growth and development. J Cell Physiol. 2019 05; 234(5):6679-6687.
Score: 0.038
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Bone marrow-derived immature myeloid cells are a main source of circulating suPAR contributing to proteinuric kidney disease. Nat Med. 2017 01; 23(1):100-106.
Score: 0.033
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CD2AP in mouse and human podocytes controls a proteolytic program that regulates cytoskeletal structure and cellular survival. J Clin Invest. 2011 Oct; 121(10):3965-80.
Score: 0.023