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Connection

Thomas Shannon to Heart Failure

This is a "connection" page, showing publications Thomas Shannon has written about Heart Failure.
Connection Strength

1.251
  1. Spontaneous Ca waves in ventricular myocytes from failing hearts depend on Ca(2+)-calmodulin-dependent protein kinase II. J Mol Cell Cardiol. 2010 Jul; 49(1):25-32.
    View in: PubMed
    Score: 0.292
  2. Ryanodine receptor Ca2+ sensitivity and excitation-contraction coupling in muscular dystrophy and heart failure: similar and yet different. Am J Physiol Heart Circ Physiol. 2009 Dec; 297(6):H1965-6.
    View in: PubMed
    Score: 0.283
  3. Diastolic release of calcium from the sarcoplasmic reticulum: a potential target for treating triggered arrhythmias and heart failure. J Am Coll Cardiol. 2009 May 26; 53(21):2006-8.
    View in: PubMed
    Score: 0.275
  4. Elevated sarcoplasmic reticulum Ca2+ leak in intact ventricular myocytes from rabbits in heart failure. Circ Res. 2003 Oct 03; 93(7):592-4.
    View in: PubMed
    Score: 0.185
  5. Intra-sarcoplasmic reticulum free [Ca2+] and buffering in arrhythmogenic failing rabbit heart. Circ Res. 2007 Oct 12; 101(8):802-10.
    View in: PubMed
    Score: 0.061
  6. Beta-adrenergic enhancement of sarcoplasmic reticulum calcium leak in cardiac myocytes is mediated by calcium/calmodulin-dependent protein kinase. Circ Res. 2007 Feb 16; 100(3):391-8.
    View in: PubMed
    Score: 0.058
  7. Ca2+/calmodulin-dependent protein kinase modulates cardiac ryanodine receptor phosphorylation and sarcoplasmic reticulum Ca2+ leak in heart failure. Circ Res. 2005 Dec 09; 97(12):1314-22.
    View in: PubMed
    Score: 0.054
  8. Quantitative assessment of the SR Ca2+ leak-load relationship. Circ Res. 2002 Oct 04; 91(7):594-600.
    View in: PubMed
    Score: 0.043
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.