PhDKathrinBanachKathrin Banach349610Banach, KathrinMy Scopus ID is 6603362447.Assistant Professorprns:coAuthorOfcoauthor ofFaculty Rankprns:fullNamefull nameprns:hasAuthorListauthor listprns:hasFacultyRankhas faculty rankprns:hasNetworkhas networkprns:hasPublicationVenuepublished inprns:informationResourceReferenceinformation resource referenceprns:isPrimaryPositionis primary positionprns:latitudelatitudeprns:longitudelongitudeprns:mainImagephotoprns:maxWeightmaximum weightprns:medlineTAjournal title abbreviationprns:meshDescriptorUIMeSH DescriptorUIprns:meshSemanticGroupNameMeSH semantic group nameprns:minWeightminimum weightprns:numberOfAuthorsnumber of authorsprns:numberOfConnectionsnumber of connectionsprns:numberOfPublicationsnumber of publicationsprns:personIdPerson IDprns:personInPrimaryPositionperson in primary positionprns:positionInDepartmentposition in departmentprns:positionInDivisionposition in divisionprns:predicateNodepredicate nodeprns:publicationDatepublication dateprns:similarTosimilar toprns:sortOrdersort orderprns:uniquenessWeightuniqueness weightprns:yearyearAcademic ArticleArticleDocumentbibo:pmidPubMed Identifierbibo:suffixNamename suffixAddressvivo:addressCitycityvivo:addressPostalCodepostal codevivo:addressStatestate or provincevivo:authorInAuthorshipselected publicationsvivo:authorRankauthor rank in publicationAuthorshipDepartmentDivisionvivo:hasResearchArearesearch areasvivo:hrJobTitleHR job titleInformation Resourcevivo:linkedAuthorlinked authorvivo:linkedInformationResourcelinked information resourcevivo:mailingAddressmailing addressvivo:overviewoverviewvivo:personInPositionpositionsPositionvivo:positionInOrganizationposition in organizationvivo:preferredTitlepreferred titlerdf:predicatepredicaterdf:typetyperdfs:labellabelConceptAgentfoaf:firstNamefirst namefoaf:lastNamelast nameOrganizationPersonD001281Disorders321950.779037Atrial FibrillationD032383Anatomy181980.818526Myocytes, CardiacD002118Chemicals & Drugs644990.72668CalciumKalipadaPahanKalipada Pahan349039Pahan, KalipadaProfessorD054462Chemicals & Drugs6120.980899p21-Activated KinasesD053595Anatomy7120.989748Embryonic Stem Cells8InstructorChicago60612IL1122selected publications3.300810.0180927174research areas1.43440.08510863coauthor of128.342.896960similar toLotharBlatterLothar Blatter41.87432800000000-87.67059170000000349611Blatter, LotharProfessorGiedriusKanaporisGiedrius Kanaporis41.87432800000000-87.67059170000000349614Kanaporis, GiedriusAssistant ProfessorEduardoRiosEduardo Rios41.87432800000000-87.67059170000000349616Rios, EduardoProfessor6Assistant Professor4Professortrue1ProfessorProfessorAuthorship 106766Authorship 130513Authorship 134556Authorship 135317Authorship 154063Authorship 155096Authorship 163803Authorship 166502Authorship 169096Authorship 169986Authorship 172896Authorship 182917Authorship 190063Authorship 190284Authorship 194603Authorship 195912Authorship 19880214586171Halbach M, Egert U, Hescheler J, Banach KCellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacologyEstimation of action potential changes from field potential recordings in multicellular mouse cardiac myocyte cultures. Cell Physiol Biochem. 2003; 13(5):271-84.Cell Physiol Biochem2003-01-01T00:00:002003Estimation of action potential changes from field potential recordings in multicellular mouse cardiac myocyte cultures.16919292Jiao Z, De Jes?s VR, Iravanian S, Campbell DP, Xu J, Vitali JA, Banach K, Fahrenbach J, Dudley SCJournal of molecular and cellular cardiologyA possible mechanism of halocarbon-induced cardiac sensitization arrhythmias. J Mol Cell Cardiol. 2006 Oct; 41(4):698-705.J Mol Cell Cardiol2006-08-17T00:00:002006A possible mechanism of halocarbon-induced cardiac sensitization arrhythmias.17092997Kapur N, Mignery GA, Banach KAmerican journal of physiology. Cell physiologyCell cycle-dependent calcium oscillations in mouse embryonic stem cells. Am J Physiol Cell Physiol. 2007 Apr; 292(4):C1510-8.Am J Physiol Cell Physiol2006-11-08T00:00:002006Cell cycle-dependent calcium oscillations in mouse embryonic stem cells.17379641Kapur N, Banach KThe Journal of physiologyInositol-1,4,5-trisphosphate-mediated spontaneous activity in mouse embryonic stem cell-derived cardiomyocytes. J Physiol. 2007 Jun 15; 581(Pt 3):1113-27.J Physiol2007-03-22T00:00:002007Inositol-1,4,5-trisphosphate-mediated spontaneous activity in mouse embryonic stem cell-derived cardiomyocytes.17901361Shang LL, Pfahnl AE, Sanyal S, Jiao Z, Allen J, Banach K, Fahrenbach J, Weiss D, Taylor WR, Zafari AM, Dudley SCCirculation researchHuman heart failure is associated with abnormal C-terminal splicing variants in the cardiac sodium channel. Circ Res. 2007 Nov 26; 101(11):1146-54.Circ Res2007-09-27T00:00:002007Human heart failure is associated with abnormal C-terminal splicing variants in the cardiac sodium channel.17932143Fahrenbach JP, Mejia-Alvarez R, Banach KThe Journal of physiologyThe relevance of non-excitable cells for cardiac pacemaker function. J Physiol. 2007 Dec 01; 585(Pt 2):565-78.J Physiol2007-10-11T00:00:002007The relevance of non-excitable cells for cardiac pacemaker function.18536930Hofgaard JP, Banach K, Mollerup S, J?rgensen HK, Olesen SP, Holstein-Rathlou NH, Nielsen MSPflugers Archiv : European journal of physiologyPhosphatidylinositol-bisphosphate regulates intercellular coupling in cardiac myocytes. Pflugers Arch. 2008 Nov; 457(2):303-13.Pflugers Arch2008-06-07T00:00:002008Phosphatidylinositol-bisphosphate regulates intercellular coupling in cardiac myocytes.18817780Fahrenbach JP, Ai X, Banach KJournal of molecular and cellular cardiologyDecreased intercellular coupling improves the function of cardiac pacemakers derived from mouse embryonic stem cells. J Mol Cell Cardiol. 2008 Nov; 45(5):642-9.J Mol Cell Cardiol2008-09-11T00:00:002008Decreased intercellular coupling improves the function of cardiac pacemakers derived from mouse embryonic stem cells.19540841Rinne A, Banach K, Blatter LAJournal of molecular and cellular cardiologyRegulation of nuclear factor of activated T cells (NFAT) in vascular endothelial cells. J Mol Cell Cardiol. 2009 Sep; 47(3):400-10.J Mol Cell Cardiol2009-06-21T00:00:002009Regulation of nuclear factor of activated T cells (NFAT) in vascular endothelial cells.20060001Grajales L, Garc?a J, Banach K, Geenen DLJournal of molecular and cellular cardiologyDelayed enrichment of mesenchymal cells promotes cardiac lineage and calcium transient development. J Mol Cell Cardiol. 2010 Apr; 48(4):735-45.J Mol Cell Cardiol2010-01-06T00:00:002010Delayed enrichment of mesenchymal cells promotes cardiac lineage and calcium transient development.20304816Rinne A, Kapur N, Molkentin JD, Pogwizd SM, Bers DM, Banach K, Blatter LAAmerican journal of physiology. Heart and circulatory physiologyIsoform- and tissue-specific regulation of the Ca(2+)-sensitive transcription factor NFAT in cardiac myocytes and heart failure. Am J Physiol Heart Circ Physiol. 2010 Jun; 298(6):H2001-9.Am J Physiol Heart Circ Physiol2010-03-19T00:00:002010Isoform- and tissue-specific regulation of the Ca(2+)-sensitive transcription factor NFAT in cardiac myocytes and heart failure.23241322Mureli S, Gans CP, Bare DJ, Geenen DL, Kumar NM, Banach KAmerican journal of physiology. Heart and circulatory physiologyMesenchymal stem cells improve cardiac conduction by upregulation of connexin 43 through paracrine signaling. Am J Physiol Heart Circ Physiol. 2013 Feb 15; 304(4):H600-9.Am J Physiol Heart Circ Physiol2012-12-15T00:00:002012Mesenchymal stem cells improve cardiac conduction by upregulation of connexin 43 through paracrine signaling.23612118DeSantiago J, Bare DJ, Ke Y, Sheehan KA, Solaro RJ, Banach KJournal of molecular and cellular cardiologyFunctional integrity of the T-tubular system in cardiomyocytes depends on p21-activated kinase 1. J Mol Cell Cardiol. 2013 Jul; 60:121-8.J Mol Cell Cardiol2013-04-20T00:00:002013Functional integrity of the T-tubular system in cardiomyocytes depends on p21-activated kinase 1.23614555DeSantiago J, Bare DJ, Banach KStem cells and developmentIschemia/Reperfusion injury protection by mesenchymal stem cell derived antioxidant capacity. Stem Cells Dev. 2013 Sep 15; 22(18):2497-507.Stem Cells Dev2013-06-11T00:00:002013Ischemia/Reperfusion injury protection by mesenchymal stem cell derived antioxidant capacity.24161911Taglieri DM, Johnson KR, Burmeister BT, Monasky MM, Spindler MJ, DeSantiago J, Banach K, Conklin BR, Carnegie GKJournal of molecular and cellular cardiologyThe C-terminus of the long AKAP13 isoform (AKAP-Lbc) is critical for development of compensatory cardiac hypertrophy. J Mol Cell Cardiol. 2014 Jan; 66:27-40.J Mol Cell Cardiol2013-10-23T00:00:002013The C-terminus of the long AKAP13 isoform (AKAP-Lbc) is critical for development of compensatory cardiac hypertrophy.24380729DeSantiago J, Bare DJ, Xiao L, Ke Y, Solaro RJ, Banach KJournal of molecular and cellular cardiologyp21-Activated kinase1 (Pak1) is a negative regulator of NADPH-oxidase 2 in ventricular myocytes. J Mol Cell Cardiol. 2014 Feb; 67:77-85.J Mol Cell Cardiol2013-12-28T00:00:002013p21-Activated kinase1 (Pak1) is a negative regulator of NADPH-oxidase 2 in ventricular myocytes.24409283Kapoor N, Maxwell JT, Mignery GA, Will D, Blatter LA, Banach KPloS oneSpatially defined InsP3-mediated signaling in embryonic stem cell-derived cardiomyocytes. PLoS One. 2014; 9(1):e83715.PLoS One2014-01-07T00:00:002014Spatially defined InsP3-mediated signaling in embryonic stem cell-derived cardiomyocytes.Neurological SciencesRush University, Rush Medical CollegeAuthorship 444374Authorship 44615534755642Varma D, Almeida JFQ, DeSantiago J, Blatter LA, Banach KJournal of molecular and cellular cardiologyInositol 1,4,5-trisphosphate receptor - reactive oxygen signaling domain regulates excitation-contraction coupling in atrial myocytes. J Mol Cell Cardiol. 2022 02; 163:147-155.J Mol Cell Cardiol2021-10-28T00:00:002021Inositol 1,4,5-trisphosphate receptor - reactive oxygen signaling domain regulates excitation-contraction coupling in atrial myocytes.35262277Dittloff KT, Spanghero E, Sol?s C, Banach K, Russell BPhysiological reportsTransthyretin deposition alters cardiomyocyte sarcomeric architecture, calcium transients, and contractile force. Physiol Rep. 2022 03; 10(5):e15207.Physiol Rep2022-03-01T00:00:002022Transthyretin deposition alters cardiomyocyte sarcomeric architecture, calcium transients, and contractile force.true1ProfessorProfessortrue1ProfessorProfessortrue1Assistant ProfessorAssistant Professortrue1ProfessorProfessorInternal Medicine, Division of CardiologyPhysiology and BiophysicsXunAiXun Ai41.87432800000000-87.67059170000000350000Ai, XunProfessortrue1Assistant ProfessorAssistant ProfessorAuthorship 460405Authorship 47954136671392Banach K, Blatter LABiomoleculesThe 'Reverse FDUF' Mechanism of Atrial Excitation-Contraction Coupling Sustains Calcium Alternans-A Hypothesis. Biomolecules. 2022 12 20; 13(1).Biomolecules2022-12-20T00:00:002022The 'Reverse FDUF' Mechanism of Atrial Excitation-Contraction Coupling Sustains Calcium Alternans-A Hypothesis.37086972Pereira CH, Bare DJ, Rosas PC, Dias FAL, Banach KJournal of molecular and cellular cardiologyThe role of P21-activated kinase (Pak1) in sinus node function. J Mol Cell Cardiol. 2023 06; 179:90-101.J Mol Cell Cardiol2023-04-20T00:00:002023The role of P21-activated kinase (Pak1) in sinus node function.true1InstructorInstructorDanBareDan Bare41.87432800000000-87.67059170000000350046Bare, DanInstructorAuthorship 32019629625277DeSantiago J, Bare DJ, Varma D, Solaro RJ, Arora R, Banach KHeart rhythmLoss of p21-activated kinase 1 (Pak1) promotes atrial arrhythmic activity. Heart Rhythm. 2018 08; 15(8):1233-1241.Heart Rhythm2018-04-03T00:00:002018Loss of p21-activated kinase 1 (Pak1) promotes atrial arrhythmic activity.