Chundo Oh to p38 Mitogen-Activated Protein Kinases
This is a "connection" page, showing publications Chundo Oh has written about p38 Mitogen-Activated Protein Kinases.
Connection Strength
0.205
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Signaling mechanisms leading to the regulation of differentiation and apoptosis of articular chondrocytes by insulin-like growth factor-1. J Biol Chem. 2003 Sep 19; 278(38):36563-71.
Score: 0.055
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Immunosuppressant rapamycin inhibits protein kinase C alpha and p38 mitogen-activated protein kinase leading to the inhibition of chondrogenesis. Eur J Pharmacol. 2001 Sep 21; 427(3):175-85.
Score: 0.049
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Opposing role of mitogen-activated protein kinase subtypes, erk-1/2 and p38, in the regulation of chondrogenesis of mesenchymes. J Biol Chem. 2000 Feb 25; 275(8):5613-9.
Score: 0.044
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p38 kinase-dependent and -independent Inhibition of protein kinase C zeta and -alpha regulates nitric oxide-induced apoptosis and dedifferentiation of articular chondrocytes. J Biol Chem. 2002 Aug 16; 277(33):30375-81.
Score: 0.013
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ERK-1/2 and p38 kinase oppositely regulate nitric oxide-induced apoptosis of chondrocytes in association with p53, caspase-3, and differentiation status. J Biol Chem. 2002 Jan 11; 277(2):1332-9.
Score: 0.012
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Epidermal growth factor negatively regulates chondrogenesis of mesenchymal cells by modulating the protein kinase C-alpha, Erk-1, and p38 MAPK signaling pathways. J Biol Chem. 2000 Apr 21; 275(16):12353-9.
Score: 0.011
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Modulation of nitric oxide-induced apoptotic death of HL-60 cells by protein kinase C and protein kinase A through mitogen-activated protein kinases and CPP32-like protease pathways. Cell Immunol. 1999 May 25; 194(1):36-46.
Score: 0.010
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Overexpression of protein kinase C isoforms protects RAW 264.7 macrophages from nitric oxide-induced apoptosis: involvement of c-Jun N-terminal kinase/stress-activated protein kinase, p38 kinase, and CPP-32 protease pathways. J Immunol. 1999 Mar 15; 162(6):3395-401.
Score: 0.010