 Glycolysis
"Glycolysis" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A metabolic process that converts GLUCOSE into two molecules of PYRUVIC ACID through a series of enzymatic reactions. Energy generated by this process is conserved in two molecules of ATP. Glycolysis is the universal catabolic pathway for glucose, free glucose, or glucose derived from complex CARBOHYDRATES, such as GLYCOGEN and STARCH.
| Descriptor ID |
D006019
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| MeSH Number(s) |
G02.111.087.160.750 G02.149.115.160.750 G03.495.256.750 G03.495.335.436 G03.495.553.360
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| Concept/Terms |
Embden-Meyerhof Pathway- Embden-Meyerhof Pathway
- Embden Meyerhof Pathway
- Embden-Meyerhof Pathways
- Pathway, Embden-Meyerhof
- Pathways, Embden-Meyerhof
- Embden-Meyerhof-Parnas Pathway
- Embden Meyerhof Parnas Pathway
- Pathway, Embden-Meyerhof-Parnas
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Below are MeSH descriptors whose meaning is more general than "Glycolysis".
Below are MeSH descriptors whose meaning is more specific than "Glycolysis".
This graph shows the total number of publications written about "Glycolysis" by people in this website by year, and whether "Glycolysis" was a major or minor topic of these publications.
To see the data from this visualization as text, click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2000 | 1 | 0 | 1 | | 2002 | 0 | 1 | 1 | | 2003 | 0 | 1 | 1 | | 2005 | 1 | 0 | 1 | | 2006 | 0 | 1 | 1 | | 2007 | 1 | 0 | 1 | | 2013 | 0 | 1 | 1 | | 2015 | 1 | 0 | 1 | | 2018 | 0 | 3 | 3 | | 2019 | 1 | 0 | 1 |
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Below are the most recent publications written about "Glycolysis" by people in Profiles.
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Zhang Z, Chen F, Li S, Guo H, Xi H, Deng J, Han Q, Zhang W. ERG the modulates Warburg effect and tumor progression in cervical cancer. Biochem Biophys Res Commun. 2020 01 29; 522(1):191-197.
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Duan L, Perez RE, Maki CG. Alpha ketoglutarate levels, regulated by p53 and OGDH, determine autophagy and cell fate/apoptosis in response to Nutlin-3a. Cancer Biol Ther. 2019; 20(3):252-260.
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Duan L, Perez RE, Chen L, Blatter LA, Maki CG. p53 promotes AKT and SP1-dependent metabolism through the pentose phosphate pathway that inhibits apoptosis in response to Nutlin-3a. J Mol Cell Biol. 2018 08 01; 10(4):331-340.
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Sikes KJ, Li J, Gao SG, Shen Q, Sandy JD, Plaas A, Wang VM. TGF-b1 or hypoxia enhance glucose metabolism and lactate production via HIF1A signaling in tendon cells. Connect Tissue Res. 2018 09; 59(5):458-471.
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Duan L, Perez RE, Davaadelger B, Dedkova EN, Blatter LA, Maki CG. p53-regulated autophagy is controlled by glycolysis and determines cell fate. Oncotarget. 2015 Sep 15; 6(27):23135-56.
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Zima AV, Pabbidi MR, Lipsius SL, Blatter LA. Effects of mitochondrial uncoupling on Ca(2+) signaling during excitation-contraction coupling in atrial myocytes. Am J Physiol Heart Circ Physiol. 2013 Apr 01; 304(7):H983-93.
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Aromolaran AS, Zima AV, Blatter LA. Role of glycolytically generated ATP for CaMKII-mediated regulation of intracellular Ca2+ signaling in bovine vascular endothelial cells. Am J Physiol Cell Physiol. 2007 Jul; 293(1):C106-18.
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Zima AV, Kockskämper J, Blatter LA. Cytosolic energy reserves determine the effect of glycolytic sugar phosphates on sarcoplasmic reticulum Ca2+ release in cat ventricular myocytes. J Physiol. 2006 Nov 15; 577(Pt 1):281-93.
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Kockskämper J, Zima AV, Blatter LA. Modulation of sarcoplasmic reticulum Ca2+ release by glycolysis in cat atrial myocytes. J Physiol. 2005 May 01; 564(Pt 3):697-714.
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Blatter LA, Kockskämper J, Sheehan KA, Zima AV, Hüser J, Lipsius SL. Local calcium gradients during excitation-contraction coupling and alternans in atrial myocytes. J Physiol. 2003 Jan 01; 546(Pt 1):19-31.
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