Lothar Blatter to Heart Atria
This is a "connection" page, showing publications Lothar Blatter has written about Heart Atria.
Connection Strength
5.785
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Mitochondrial calcium uniporter complex activation protects against calcium alternans in atrial myocytes. Am J Physiol Heart Circ Physiol. 2020 10 01; 319(4):H873-H881.
Score: 0.695
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Action potential shortening rescues atrial calcium alternans. J Physiol. 2019 02; 597(3):723-740.
Score: 0.617
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The intricacies of atrial calcium cycling during excitation-contraction coupling. J Gen Physiol. 2017 09 04; 149(9):857-865.
Score: 0.563
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Alternans in atria: Mechanisms and clinical relevance. Medicina (Kaunas). 2017; 53(3):139-149.
Score: 0.556
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A novel mechanism of tandem activation of ryanodine receptors by cytosolic and SR luminal Ca2+ during excitation-contraction coupling in atrial myocytes. J Physiol. 2017 06 15; 595(12):3835-3845.
Score: 0.543
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Inositol-1,4,5-trisphosphate induced Ca2+ release and excitation-contraction coupling in atrial myocytes from normal and failing hearts. J Physiol. 2015 Mar 15; 593(6):1459-77.
Score: 0.469
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Modulation of sarcoplasmic reticulum Ca2+ release by glycolysis in cat atrial myocytes. J Physiol. 2005 May 01; 564(Pt 3):697-714.
Score: 0.236
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Increased Risk for Atrial Alternans in Rabbit Heart Failure: The Role of Ca2+/Calmodulin-Dependent Kinase II and Inositol-1,4,5-trisphosphate Signaling. Biomolecules. 2023 Dec 30; 14(1).
Score: 0.219
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Calcium- and voltage-driven atrial alternans: Insight from [Ca]i and Vm asynchrony. Physiol Rep. 2023 05; 11(10):e15703.
Score: 0.209
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The 'Reverse FDUF' Mechanism of Atrial Excitation-Contraction Coupling Sustains Calcium Alternans-A Hypothesis. Biomolecules. 2022 12 20; 13(1).
Score: 0.204
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Membrane potential determines calcium alternans through modulation of SR Ca2+ load and L-type Ca2+ current. J Mol Cell Cardiol. 2017 04; 105:49-58.
Score: 0.136
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Dyssynchronous calcium removal in heart failure-induced atrial remodeling. Am J Physiol Heart Circ Physiol. 2016 12 01; 311(6):H1352-H1359.
Score: 0.133
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Calcium gradients during excitation-contraction coupling in cat atrial myocytes. J Physiol. 1996 Aug 01; 494 ( Pt 3):641-51.
Score: 0.131
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Calcium-activated chloride current determines action potential morphology during calcium alternans in atrial myocytes. J Physiol. 2016 Feb 01; 594(3):699-714.
Score: 0.126
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The mechanisms of calcium cycling and action potential dynamics in cardiac alternans. Circ Res. 2015 Feb 27; 116(5):846-56.
Score: 0.117
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Effects of mitochondrial uncoupling on Ca(2+) signaling during excitation-contraction coupling in atrial myocytes. Am J Physiol Heart Circ Physiol. 2013 Apr 01; 304(7):H983-93.
Score: 0.103
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Regulation of cardiac alternans by ?-adrenergic signaling pathways. Am J Physiol Heart Circ Physiol. 2012 Oct 15; 303(8):H1047-56.
Score: 0.100
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Isoform- and tissue-specific regulation of the Ca(2+)-sensitive transcription factor NFAT in cardiac myocytes and heart failure. Am J Physiol Heart Circ Physiol. 2010 Jun; 298(6):H2001-9.
Score: 0.084
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IP3-dependent nuclear Ca2+ signalling in the mammalian heart. J Physiol. 2007 Oct 15; 584(Pt 2):601-11.
Score: 0.071
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Regional differences in spontaneous Ca2+ spark activity and regulation in cat atrial myocytes. J Physiol. 2006 May 01; 572(Pt 3):799-809.
Score: 0.064
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Endothelin-1-induced arrhythmogenic Ca2+ signaling is abolished in atrial myocytes of inositol-1,4,5-trisphosphate(IP3)-receptor type 2-deficient mice. Circ Res. 2005 Jun 24; 96(12):1274-81.
Score: 0.060
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Palytoxin disrupts cardiac excitation-contraction coupling through interactions with P-type ion pumps. Am J Physiol Cell Physiol. 2004 Aug; 287(2):C527-38.
Score: 0.056
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Inositol-1,4,5-trisphosphate-dependent Ca(2+) signalling in cat atrial excitation-contraction coupling and arrhythmias. J Physiol. 2004 Mar 16; 555(Pt 3):607-15.
Score: 0.055
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Subcellular Ca2+ alternans represents a novel mechanism for the generation of arrhythmogenic Ca2+ waves in cat atrial myocytes. J Physiol. 2002 11 15; 545(1):65-79.
Score: 0.051
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Functional coupling between glycolysis and excitation-contraction coupling underlies alternans in cat heart cells. J Physiol. 2000 May 01; 524 Pt 3:795-806.
Score: 0.042
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The role of fibroblast - Cardiomyocyte interaction for atrial dysfunction in HFpEF and hypertensive heart disease. J Mol Cell Cardiol. 2019 06; 131:53-65.
Score: 0.040
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Ginsenoside Re suppresses electromechanical alternans in cat and human cardiomyocytes. Am J Physiol Heart Circ Physiol. 2008 Aug; 295(2):H851-9.
Score: 0.019
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Phenylephrine acts via IP3-dependent intracellular NO release to stimulate L-type Ca2+ current in cat atrial myocytes. J Physiol. 2005 Aug 15; 567(Pt 1):143-57.
Score: 0.015
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Signaling mechanisms that mediate nitric oxide production induced by acetylcholine exposure and withdrawal in cat atrial myocytes. Circ Res. 2003 Dec 12; 93(12):1233-40.
Score: 0.014
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Acute exposure to thyroid hormone increases Na+ current and intracellular Ca2+ in cat atrial myocytes. J Physiol. 2003 Jan 15; 546(Pt 2):491-9.
Score: 0.013
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Nitric oxide signalling by selective beta(2)-adrenoceptor stimulation prevents ACh-induced inhibition of beta(2)-stimulated Ca(2+) current in cat atrial myocytes. J Physiol. 2002 Aug 01; 542(Pt 3):711-23.
Score: 0.012
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Beta 2-adrenergic receptor signaling acts via NO release to mediate ACh-induced activation of ATP-sensitive K+ current in cat atrial myocytes. J Gen Physiol. 2002 Jan; 119(1):69-82.
Score: 0.012
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Intracellular Ca2+ release contributes to automaticity in cat atrial pacemaker cells. J Physiol. 2000 Apr 15; 524 Pt 2:415-22.
Score: 0.011
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Withdrawal of acetylcholine elicits Ca2+-induced delayed afterdepolarizations in cat atrial myocytes. Circulation. 1997 Aug 19; 96(4):1275-81.
Score: 0.009